在 中文 中使用 髓鞘 的示例及其翻译为 英语
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大脑发育的第五个过程是髓鞘作用。
要研究髓鞘，你真的需要研究这两种细胞。
其结果是，再次，髓鞘损失和神经信号传导不良。
The result is, again, a loss of myelin and poor nerve signal transmission.它会破坏髓鞘，这是围绕一些神经细胞的保护性表面。
It destroys myelin, the protective surface that surrounds some nerve cells.当髓鞘不再完整时，神经细胞之间的通讯受损。
When the myelin sheath is no longer intact, communication between nerve cells is impaired.当轴突被髓鞘包裹时，称为轴突神经纤维。
When the axon is covered with a sheath, it is called nerve fibre.Leu-7和髓鞘碱性蛋白亦分别见于50％和40％的病例。
Leu-7 and myelin basic protein are noted in 50% and 40% of cases respectively.您现在也可以很容易地在酶IHC染色髓鞘，使用LuxOL快速蓝色特殊染色试剂盒。
You can now also easily stain myelin in enzyme IHC, using the Luxol fast blue special stain kit.可能参与髓鞘的完成和/或维持，并参与细胞-细胞的通讯。
May be involved in completion and/or maintenance of the myelin sheath and in cell-cell communication.随着时间的推移，这种压力会导致周围神经的保护外衣，称为髓鞘，拖垮。
Over time, this pressure will cause the protective coat around the nerve,called a myelin sheath, to wear down.以前，这种髓鞘追踪将需要几个月到几年的人工注释执行。
Previously, this kind of myelin tracing would have required months to years for human annotators to perform.功能性间隙连接的丧失可能损害雪旺氏细胞的正常活性，例如髓鞘生成。
The loss of functional gap junctions probably impairs the normal activity of Schwann cells,such as myelin production.MyelinBasicProtein(MBP)(68-82),guineapig是髓鞘碱性蛋白(MBP)的一个片段。
Myelin Basic Protein(MBP)(68-82), guinea pig is a fragment of myelin basic protein(MBP).神经元与髓鞘出现白色的，而不是灰色，所以我们分享我们的大脑无论在"白色"和"灰色"。
Neurons with a myelin sheath appear white rather than grey, so we share our brain matter on the"white" and"gray".髓鞘正常化后，我们发现突变小鼠的社会行为也正常化了，”Barak博士说。
After the myelin sheaths normalized, we found that the mutant mice's social behavior normalized as well," Dr. Barak says.CMT3也可由位于染色体1上的MPZ基因中的突变引起，所述突变编码髓鞘蛋白零。
CMT3 can also be caused by mutations in the MPZ gene located on chromosome 1,which encodes for the myelin protein zero.当髓鞘受损时，信息不再能够从大脑和脊髓清晰地传递到身体的其他部位。
When myelin is damaged, messages can no longer be clearly transmitted from the brain and spinal cord to other parts of the body.因此，有更少的炎性细胞进入中枢神经系统（CNS）损害髓鞘。
Therefore, there are less inflammatory cells available to cross into the central nervous system(CNS)where they could damage myelin.有缺陷的EGR2蛋白不能与DNA结合，导致髓鞘损失和神经信号传递受损。
The defective EGR2 protein is unable to bind to the DNA,resulting in the loss of myelin and impaired nerve signal transmission.虽然每种成像技术都提供有价值的信息，但他们自己只画出髓鞘厚度的模糊肖像。
While each imaging technique provides valuable information,by themselves they paint only a hazy portrait of myelin thickness.尽管每种成像技术都提供了有价值的信息，但是它们本身只绘制了朦胧的髓鞘厚度。
While each imaging technique provides valuable information,by themselves they paint only a hazy portrait of myelin thickness.该研究生成的新知识可以更深入地阐明影响髓鞘的一些疾病，例如多发性硬化症（MS）。
The new knowledge the project has generatedcan shed more light on diseases that affect the myelin, such as multiple sclerosis(MS).没有髓鞘的话，神经元的电信号传送就不佳，部分细胞由于缺乏少突胶质细胞代谢支持而死亡。
Without myelin, the electrical signals sent by neurons travel poorly and some cells die due to the lack of metabolic support from oligodendrocytes.EGR2中的突变导致异常蛋白质不能结合到DNA上，从而阻止了参与髓鞘生成的基因的表达。
Mutations in EGR2 result in an abnormal protein that is unable to bind to the DNA,preventing the expression of genes involved in myelin production.修复中枢神经系统中的髓鞘损伤没有选择性的压力，因为你可能会死亡，”Monk说。
In contrast,“There's no selective pressure in repairing myelin damage in the central nervous system, because you're probably going to die,” Monk explained.当他们敲除转基因小鼠的该基因后，出现了一个意想不到的现象：单个施万细胞开始在多个轴突上铺展髓鞘。
When they knocked out the gene in genetically modified mice, they discovered an unexpected characteristic:individual Schwann cells began spreading myelin across many axons.最后，脂肪也用于脂肪髓鞘的形成，充当神经细胞周围的物质，改善神经传导的速度和效率。
Finally, fat is also used in the formation of myelin, a substance surrounding nerve cells that improves the speed and efficiency 据认为，在复发缓解型多发性硬化症（RRMS）患者中，ponesimod可防止炎性免疫细胞越过血脑屏障进入中枢神经系统（CNS）和损害髓鞘。
It is thought that in people with relapsing-remitting multiple sclerosis(RRMS),ponesimod prevents immune cells from crossing the blood-brain barrier and damaging myelin.如果我们了解如何控制髓鞘异常，也许我们的理解可以帮助WS、MS甚至某些自闭症患者，”Barak博士总结道。
If we understand how to control myelination abnormalities, perhaps our understanding can help WS, MS and even certain autism patients," Dr. Barak concludes.此外，在整个成人期，我们都保持着髓鞘生长的能力，这是一种绝缘鞘，可以保护神经元以及在它们之间传导的速度信号。
What's more, throughout adulthood, we maintain the ability to grow myelin, a sort of insulating sheath that protects neurons and speeds signals traveling between them.
