Приклади вживання Elegans Англійська мовою та їх переклад на Українською
{-}
-
Colloquial
-
Ecclesiastic
-
Computer
Browsing by Keywords"C. elegans".
Of course, C. elegans are a far cry from humans.
On the other side, there is C. elegans.
Hypericum elegans. Photo by: Konstantin Teplov.
The discovery was made during experiments with the worm Caenorhabditis elegans.
Elegans, scientists have found, you can say"aging molecule".
The Aurora B complex is necessary for cytokinesis in vertebrates, C. elegans, Drosophila, and fission yeast.
Elegans can rightly be considered one of the most delicious host varieties.
The Rhabditidae are a family of nematodes which includes the model organism Caenorhabditis elegans.
Elegans was the first multicellular organism whose genome was completely sequenced.
The multicellular animals, Caenorhabditis elegans, are ideal as a system to address the above questions.
Elegans, the worms not only aged slower, but they also stayed healthier longer.
Scientists at Michigan State University found that the brain andthe intestinal in a worm called Caenorhabditis elegans control the aging process.
Similarly, in C. elegans, condensin activity is dependent on Aurora B in metaphase.
With a typical lifespan of just three to four weeks, there's a chance you havemilk in your fridge older than the average C. elegans worm.
Elegans live for about 21 days, and somewhere in the 10-11 day there is a change in behavior.
During the experiment on the worm Caenorhabditis elegans has been established that free radicals are involved in programmed cell death(apoptosis).
Elegans have a biochemical environment similar to that of humans and are a popular research tool for the study of the biology of ageing and are used to model human diseases.
For instance, mammals have a meiosis-specific variant of SMC1, known as SMC1β.[10]The nematode Caenorhabditis elegans has an SMC4-variant that has a specialized role in dosage compensation.[11].
The nematode Caenorhabditis elegans has an SMC4-variant that has a specialized role in dosage compensation.
The CRISPR repeats are conserved amongst many species and have been demonstrated to be usable in human cells,[11] bacteria,[12]C. elegans,[13] zebrafish,[14] and other organisms for effective genome manipulation.
Particular pride hosts Elegans- large heart-shaped jade leaves, the length of which can reach 37 cm.
In the course of the study, experts found that increasing or decreasing the concentration of Kruppel-like factors(KLFs)regulating gene activity allows for the prolonging or shortening of the life of Caenorhabditis elegans nematodes.
Dauer formation in C. elegans requires a nuclear receptor DAF-12 and a forkhead transcription factor daf-16.
Specifically, Aurora B localizes to the chromosomes in prophase, the centromere in prometaphase and metaphase, and the central mitotic spindle in anaphase.[4] This localization has been determinedby indirect immunofluorescence in mammalian, C. elegans, and Drosophila cells.
Elegans has a biochemical environment and cellular properties similar to that of humans and is a popular research tool for studying the biology of aging and as a model of human disease.
Blue-and-white swallow, Pygochelidon cyanoleuca Black-collared swallow, Pygochelidon melanoleuca Tawny-headed swallow, Alopochelidon fucata Andean swallow, Orochelidon andecola Southern rough-winged swallow, Stelgidopteryx ruficollis Brown-chested martin, Progne tapera Purple martin, Progne subis(V)Gray-breasted martin, Progne chalybea Southern martin, Progne elegans White-winged swallow, Tachycineta albiventer White-rumped swallow, Tachycineta leucorrhoa Chilean swallow, Tachycineta leucopyga Bank swallow, Riparia riparia Barn swallow, Hirundo rustica Cliff swallow, Petrochelidon pyrrhonota.
Elegans and human pathogens, in the second/third year of the project, the student may have the opportunity to examine the molecular underpinnings of evolved worm host resistance to different pathogens.
The nematode Caenorhabditis elegans possesses a third complex(closely related to condensin I) that participates in chromosome-wide gene regulation, i.e., dosage compensation.[7] In this complex, known as condensin IDC, the authentic SMC4 subunit is replaced with its variant, DPY-27.
Similarly, in C. elegans, condensin activity is dependent on Aurora B in metaphase.[20] However, in Xenopus egg cell-free extracts, condensin binding and chromosome condensation occur normally even in the absence of Aurora B.[21] Likewise, after treating cells with an Aurora B enzyme inhibitor(Aurora B localization is not affected), the condensin complex localizes normally.