Примери за използване на Beta-amyloid на Английски и техните преводи на Български
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Scan with moderate beta-amyloid deposition.
Beta-amyloid is a protein that is characteristic of Alzheimer's disease(AD).
There has been evidence that beta-amyloid may spread like prions.
Structure of human insulin-degrading enzyme(IDE)in complex with beta-amyloid.
Beta-amyloid is a peptide that is involved in the development of Alzheimer's disease.
At first, the healthy mice started to accumulate beta-amyloid in their brains.
Many researchers believe that beta-amyloid is a key player in this memory-destroying disease.
Inhibits pro-inflammatory cytokines anddecreases pigmentation of beta-amyloid.
There has been evidence that beta-amyloid may spread like prions.
Like CJD, Alzheimer's also involves a misfolded protein called beta-amyloid.
Specifically, there is a buildup of beta-amyloid, which is a protein that produces amyloid plaques.
Alzheimer's disease stoppable much earlier by reducing beta-amyloid protein.
And physical activity also decreases beta-amyloid proteins, leading to less disruption between brain neurons. Well.
Pterostilbene powder may prevent Alzheimer's disease by protecting the brain from beta-amyloid(Aβ).
Plaques, deposits of a protein fragment called beta-amyloid, look like clumps in the spaces between neurons.
The scientists carried out their research using mice that develop pathological aggregates of tau and beta-amyloid.
In turn, these antibodies reduced levels of the insoluble tau and beta-amyloid that produce plaques and tangles.
These include proteins called beta-amyloid and tau, found in the spinal fluid during the early stages of the disease.
They established a connection between daytime sleepiness and beta-amyloid accumulation in the brain.
Scientists believe that the combination of beta-amyloid plaques and neurofibrillary tangles might work together to speed up neurodegeneration.
But animals in which AHN was induced by exercise showed improved cognitive performance andreduced levels of beta-amyloid.
Plaque is characterized by deposits of a toxic protein called beta-amyloid in the spaces between nerve cells in the brain.
Beta-amyloid also appears to impair brain function by destroying synapses(structures through which nerve cells transmit signals to one another).
One of those waste products is a protein called beta-amyloid, which contributes to the development of Alzheimer's disease14.
It is the J147 study that has led scientists to discover that endocannabinoids are involved in the elimination of beta-amyloid and the reduction of inflammation.
It has been found to inhibit the formation of beta-amyloid fibril, which means that it is a very powerful aid for people suffering from Alzheimer's and dementia.
Disorders in the treatment of the molecules of this protein lead to the appearance of beta-amyloid plaques and the destruction of nerve cells.
These clumps of a protein called beta-amyloid may damage and destroy brain cells in several ways, including interfering with cell-to-cell communication.
Plaques are found between the dying cells in the brain- from the build-up of a protein called beta-amyloid(you may hear the term“amyloid plaques”).
It has long been known that beta-amyloid accumulates in nerve cells aging brain well before the onset of symptoms and plaques of Alzheimer's disease.