Примери за използване на Fasting plasma glucose на Английски и техните преводи на Български
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Fasting plasma glucose and insulin are determinants of dietary weight loss success.
Dose-adjustments are recommended to be based on fasting plasma glucose measurements.
It may have a beneficial effect on fasting plasma glucose, LDL cholesterol, HDL cholesterol, and triglyceride levels in patients with type 2 diabetes.
It is recommended to optimise glycaemic control via dose adjustment based on fasting plasma glucose.
Fasting plasma glucose levels were significantly lower in patients treated with EXUBERA regimens compared with those treated with subcutaneous insulin.
Both treatment arms were titrated to achieve target fasting plasma glucose levels(see section 5.1).
Diabetes' diagnostic criterion is fasting plasma glucose≥ 7.0 mmol/L which is a diagnostic point chosen because of micro-vascular issues such as diabetes retinotherapy.
Increased glucose production by the liver is the primary disorder leading to elevated fasting plasma glucose.
The majority of the observed reduction in fasting plasma glucose occurs after a single dose, consistent with the pharmacokinetic profile of albiglutide.
They were also assessed for diabetes using common laboratory measurements including fasting plasma glucose and haemoglobin A1c.
It demonstrates superior improvements in fasting plasma glucose levels compared to patients treated with BIAsp 30.
It is recommended to optimise glycaemic control via dose adjustment based on fasting plasma glucose(see section 5.1).
In combination with metformin, which mainly affected fasting plasma glucose, the effect of nateglinide on HbA1c was additive compared to either agent alone.
Participants of the study were also assessed for diabetes with the help of common lab measurements including fasting plasma glucose and hemoglobin A1c.
The diagnostic criterion for diabetes is fasting plasma glucose≥ 7.0 mmol/L- a diagnostic point selected on the basis of micro-vascular complications such as diabetic retinopathy.
Treatment with 25 mg alogliptin once daily resulted in statistically significant improvements from baseline in HbA1c and fasting plasma glucose compared to placebo-control at Week 26(Table 2).
Fasting plasma glucose levels were significantly lower in patients treated with regimens including EXUBERA compared with those treated with subcutaneously administered fast-acting human insulin only regimens.
This was determinedby clinically relevant and statistically significant reductions in glycosylated haemoglobin(HbA1c) and fasting plasma glucose compared to control from baseline to study endpoint.
The reductions in fasting plasma glucose obtained with Lyxumia treatment ranged from 0.42 mmol/L to 1.19 mmol/L(7.6 to 21.4 mg/dl) from baseline at the end of the main 24-week treatment period in placebo-controlled studies.
In comparative studies with sulfonylurea preparation, pioglitazone leads to a similar reduction in HbA1c and fasting plasma glucose. The effect is slower and more prolonged than the earlier effect of the sulfonylurea medicine.
After measuring fasting plasma glucose, insulin, cholesterol, triglycerides, exhaled(breath) nitric oxide metabolites and plasma levels before and after the study, subjects on average experienced improvements in fasting glucose and insulin levels, a reduction in total cholesterol and borderline significant reduction in LDL-cholesterol levels.
In order for the researchers to confirm the accuracy of their protocol,subjects also returned for another blood test that measured fasting plasma glucose to confirm whether or not they had diabetes or pre-diabetes.
Importantly, despite alogliptin and glipizide having similar HbA1c and fasting plasma glucose changes from baseline, episodes of hypoglycaemia were notably less frequent in patients receiving 25 mg alogliptin(5.4%) compared to those receiving glipizide(26.0%).
Treatment with dapagliflozin plus metformin at all doses produced clinically relevant andstatistically significant improvements in HbA1c and fasting plasma glucose compared with placebo in combination with metformin.
Treatment with saxagliptin 5 mg once daily produced clinically relevant andstatistically significant improvements in haemoglobin A1c(HbA1c), fasting plasma glucose(FPG) and postprandial glucose(PPG) compared to placebo in monotherapy, in combination with metformin(initial or add-on therapy), in combination with a sulphonylurea, and in combination with a thiazolidinedione(see Table 2).
Alogliptin as add-on therapy to insulin(with or without metformin) The addition of 25 mg alogliptin once daily to insulin therapy(mean dose= 56.5 IU, with or without metformin)resulted in statistically significant improvements from baseline in HbA1c and fasting plasma glucose at Week 26 when compared to the addition of placebo(Table 2).
In patients with type 1 diabetes mellitus, treatment with Ryzodeg o.d. plus IAsp for the remaining meals demonstrated similar glycaemic control(HbA1c and fasting plasma glucose) with a lower rate of nocturnal hypoglycaemia compared to a basal/bolus regimen with IDet plus IAsp at all meals Table.
Alogliptin as add-on therapy to a thiazolidinedione The addition of 25 mg alogliptin once daily to pioglitazone therapy(mean dose= 35.0 mg, with or without metformin or a sulphonylurea)resulted in statistically significant improvements from baseline in HbA1c and fasting plasma glucose at Week 26 when compared to the addition of placebo(Table 2).
Similar effects on glycohaemoglobin and the incidence of symptomatic hypoglycaemia were observedin both treatment groups, however fasting plasma glucose decreased more from baseline in the insulin glargine group than in the NPH group.
Treatment with saxagliptin 5 mg once daily produced clinically relevant andstatistically significant improvements in haemoglobin A1c(HbA1c), fasting plasma glucose(FPG) and postprandial glucose(PPG) compared to placebo in combination with metformin(initial or add-on therapy).