Примери за използване на Gastric acid secretion на Английски и техните преводи на Български
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Effect on gastric acid secretion.
It has a powerful andsustained suppressant effect on gastric acid secretion.
Suppression of gastric acid secretion might result in lower exposure to nilotinib.
Rivastigmine may cause increased gastric acid secretions.
Long-term suppression of gastric acid secretion by H2 antagonists or proton pump inhibitors(e.g. famotidine and omeprazole) is likely to reduce dasatinib exposure.
Rivastigmine may cause increased gastric acid secretions.
Lansoprazole is a proton pump inhibitor that inhibits gastric acid secretion and is used for the treatment of duodenal and benign gastric ulcers, gastro- oesophageal reflux disease and associated conditions.
The major metabolites of esomeprazole have no effect on gastric acid secretion.
Co-administration of Iclusig with a potent inhibitor of gastric acid secretion resulted in a minor reduction in ponatinib Cmax without a reduction in AUC0-∞.
No metabolite has been found to have any effect on gastric acid secretion.
However, when there is no food in the stomach andthere is an increased gastric acid secretion due to certain reasons, for example due to stress, it comes to damage of the lining of the stomach.
There is a physiologic balance in normal conditions between gastroduodenal mucosal defense and gastric acid secretion.
The reason being,caffeine causes increase in gastric acid secretion, which can have a laxative effect.
Under normal conditions, a physiologic balance exists between gastroduodenal mucosal defense and gastric acid secretion.
Omeprazole, a racemic mixture of two enantiomers reduces gastric acid secretion through a highly targeted mechanism of action.
Nilotinib has pH dependent solubility, therefore,absorption of nilotinib might be reduced by substances that suppress gastric acid secretion.
Esomeprazole is the S-isomer of omeprazole and reduces gastric acid secretion through a specific targeted mechanism of action.
These gastric effects in the rat are the result of sustained, pronounced hypergastrinaemia secondary to reduced production of gastric acid andare observed after long-term treatment in the rat with inhibitors of gastric acid secretion.
Helps maintain proper body weight,controls the amount of gastric acid secretion and lowers cholesterol and triglycerides in the blood.
The naturally occurring human glucagon-like peptide-2(GLP-2) is a peptide secreted by L cells of the intestine which is known to increase intestinal and portal blood flow,inhibit gastric acid secretion, and decrease intestinal motility.
Peptic ulcer disease is often treated by decreasing or eliminating gastric acid secretion, and such treatment blocks the protective antibacterial action of gastric uid.
Thereafter, the dose can be titrated up ordown as needed using measurements of gastric acid secretion to guide.
The treatment contains lifestyle modifications and control of gastric acid secretion through medical therapy with antacids or proton pump inhibitors or surgical treatment with corrective antireflux surgery.
Intravenous omeprazole produces a dose dependent inhibition of gastric acid secretion in humans.
Lansoprazole is a proton pump inhibitor that inhibits gastric acid secretion and is used for the treatment of duodenal and benign gastric ulcers, gastro-oesophageal reflux disease and associated conditions.
It is rapidly acting andprovides control through reversible inhibition of gastric acid secretion with once daily dosing.
Peripheral antimuscarinic effects that are produced as the dose increases are:decreased production of secretions from the salivary, bronchial and sweat glands; dilatation of the pupils(mydriasis) and paralysis of accommodation(cyclopegia); increased heart rate; inhibition of micturition and reduction in gastrointestinal tone; inhibition of gastric acid secretion.
After surgery or some procedures, proton pump inhibitors(PPIs)may help prevent gastric acid secretion, and this can prevent reflux.
This medicine may increase the risk for serious intestinal disease with bloody stools(necrotising enterocolitis) when administered with medicines used to treat gastric disease(such as antihistamine H2 receptor blockers orproton-pump inhibitors that reduces gastric acid secretion).
A health care provider treats Zollinger-Ellison syndrome with medications to reduce gastric acid secretion and with surgery to remove gastrinomas.