Примери за използване на Nicotinamide adenine на Английски и техните преводи на Български
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Deficiency in NADPH(nicotinamide adenine dinucleotide phosphate) reductase.
This bioactive nucleotide is an intermediate in the biosynthesis of nicotinamide adenine dinucleotide or NAD+.
Description Nicotinamide adenine dinucleotide(NADH) is the active coenzyme form of.
NMN is an intermediate in the biosynthesis of Nicotinamide adenine dinucleotide(NAD+).
Nicotinamide adenine dinucleotide(NAD) is a coenzyme found in all living cells.
The first pathway results in the formation of nicotinamide adenine dinucleotide(NAD) and nicotinamide. .
Nicotinamide adenine dinucleotide exists in two forms, an oxidized and reduced form abbreviated as NAD+ and NADH respectively.
One of the very few promising treatments for Alzheimer's is the coenzyme, NADH(nicotinamide adenine dinucleotide)[12].
NADPH, or reduced nicotinamide adenine dinucleotide phosphate, is used as a reductive reservoir of electrons to recharge antioxidants once they become oxidized.
A 1,000 mg daily of NR boosted levels of another compound called nicotinamide adenine dinucleotide(NAD+) by 60 per cent.
Nicotinamide Adenine Dinucleotide is a coeznyme consisting of an adenine base and a nicotinamide base connected by a pair of bridging phosphate group.
One of the key players involved in ATP production is the redox molecule Nicotinamide adenine dinucleotide(NAD).
Take, for example, the levels of nicotinamide adenine dinucleotide decreases in multiple organs, leading to a decline in energy production by the cell's mitochondrion.
The researchers found that 1,000 mg daily of NR boosted levels of another compound called nicotinamide adenine dinucleotide(NAD+) by 60 percent.
Importantly, it will also reduce nicotinamide adenine dinucleotide phosphate(NADPH), which is essentially the true cellular battery of your cell and has the reductive potential to recharge your antioxidants.
A 1,000 mg dosage of nicotinamide riboside a day boosted levels of another compound known as nicotinamide adenine dinucleotide(NAD+) by 60%.
It's the glue that makes ATP by helping oxygen(O)bind with nicotinamide adenine dinucleotide(NAD) and hydrogen(H) to form NADH, the hydrogen ions that make ATP synthase.
The study showed that taking 1,000 mg daily of NR can boost your levels of another compound called nicotinamide adenine dinucleotide(NAD+) by 60%.
In cells, niacin is incorporated into nicotinamide adenine dinucleotide(NAD) and nicotinamide adenine dinucleotide phosphate(NADP), although the pathways for nicotinamide and nicotinic acid are very similar.
The results showed that taking 1,000 milligrams of nicotinamide riboside each day increased levels of nicotinamide adenine dinucleotide(NAD+) by 60 percent.
Niacin is the precursor for two cofactors, NAD(nicotinamide adenine dinucleotide) and NADP(nicotinamide adenine phosphate dinucleotide), which are essential for the operation of a wide range of enzymes involved in redox reactions.
No one really thought much about it until scientists discovered that our bodies convert NR into something called nicotinamide adenine dinucleotide(NAD+).
Over the years, the study andthe therapeutic uses of nicotinamide adenine dinucleotide and its precursors have been all the rage.
Current research states it may be possible to reverse mitochondrial decay with dietary supplements that boost cellular levels of a molecule called NAD(Nicotinamide Adenine Dinucleotide).
Two unique forms of vitamin B3- nicotinamide adenine dinucleotide and nicotinamide adenine dinucleotide phosphate are essential to making the information contained in the body protein, fat and carbohydrates into usable energy.
The researchers traced the cause of a number of abnormal embryonic developments to a deficiency in nicotinamide adenine dinucleotide(NAD), a molecule that plays a key role in metabolic regulation.
Two unique forms of vitamin B3(called nicotinamide adenine dinucleotide, or NAD and nicotinamide adenine dinucleotide phosphate, or NADP) are essential for conversion of dietary proteins, fats and carbohydrates into usable energy.
The main metabolic pathway for tegafur is through conversion to 5-FU via CYP2A6 in the liver, whereas gimeracil was stable in human liver homogenate(S9 fraction) with adenosine 3'-phosphate 5'-phosphosulfphate lithium salt(PAPS;a co-factor for sulfotransferase) or nicotinamide adenine dinucleotide phosphate(NADPH).