Примери за използване на Oxidative phosphorylation на Английски и техните преводи на Български
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Then finally Oxidative phosphorylation.
This process of generating ATP this way is called oxidative phosphorylation.
Here, the superoxide released by processes such as oxidative phosphorylation is first converted to hydrogen peroxide and then further reduced to give water.
This bacterium is able to synthesize ATP by oxidative phosphorylation.
It is also a cofactor andcoenzyme in mitochondrial oxidative phosphorylation, which lowers blood pressure and is often low in those with high blood pressure(28).
Mitochondrial diseases caused by defects in the oxidative phosphorylation.
In oxidative phosphorylation, electrons are transferred from a low-energy electron donor e.g., NADctron acceptor(e.g., O2) through an electron transport chain.
Magnesium is required for energy production, oxidative phosphorylation and glycolysis.
These genes are fundamental for the correct synthesis of the cytochrome C oxidase(or complex IV)enzyme involved in the process of oxidative phosphorylation.
Magnesium is required for energy production, oxidative phosphorylation and glycolysis.
In oxidative phosphorylation, electrons are transferred from a high-energy electron donor(e.g., NADH) to an electron acceptor(e.g., O2) through an electron transport chain.
It is also important for energy production, oxidative phosphorylation, and glycolysis.
The synthesis of ATP, operated by the mitochondria, is a very complex process,which biologists identify with the term oxidative phosphorylation.
Cytochrome C, which takes an active part in the oxidative phosphorylation(the main way of producing energy with high efficiency);
It repairs possible damages on the electron transport chain and improves the oxidative phosphorylation.
Thermogenic pills use the process of oxidative phosphorylation to make the body use calories more quickly when exercising and to stabilize your body temperature.
At the time, the biochemical mechanism of ATP synthesis by oxidative phosphorylation was unknown.
It blocks oxidative phosphorylation in the cartilaginous(and renal) mitochondria, by diffusion from the inner membrane region as a proton carrier back to the mitochondrial space where it is again ionized to release protons.
Mitchell's chemiosmotic hypothesis was the basis for understanding the actual process of oxidative phosphorylation.
Mitochondrial diseases caused by defects in electron transport and oxidative phosphorylation, different genetic heterogeneity, which is due to….
The primary biochemical role of FADH2 in eukaryotes is to carry high-energy electrons used for oxidative phosphorylation.
Magnesium serves several important functions in the human body; it is a co-factor for DNA and protein synthesis, oxidative phosphorylation, enzyme activity and regulation of parathyroid hormone secretion(Volpe, 2013; Ayuk& Gittoes, 2011; Shils et al, 1999).
Mitochondrial DNA produces enzymes(ie proteins),necessary for the correct realization of the delicate process of oxidative phosphorylation.
It uncouples oxidative phosphorylation in cartilaginous(and hepatic) mitochondria, by diffusing from the inner membrane space as a proton carrier back into the mitochondrial matrix, where it ionizes once again to release protons.
The primary biochemical role of FADH 2 is to carry high-energy electrons used for oxidative phosphorylation of ADP to form ATP.
These genes contain the information that leads to the synthesis of the so-called complex I(or NADH oxide-reductase),one of the various enzymes involved in the process of oxidative phosphorylation.
Oligomycin and other substances that bind to the phosphate component of the ATP-synthase act as inhibitors of oxidative phosphorylation because these terminate the inverse transfer of protons from the intramembranous space to the matrix in the mitochondria.
This protein acts as a protector of cancer cells,shielding them from oxidative stress by inhibiting mitochondrial oxidative phosphorylation.
The substance is highly polar, hydrophilic andtransport appears to be an active process closely linked to electron transport, oxidative phosphorylation and the respiratory quinones in the cell membrane.
Secondly, mutations in mitochondrial DNA affect only certain tissues in the body,particularly those that require large amounts of ATP resulting from the process of oxidative phosphorylation.