Примери за използване на Steatohepatitis на Английски и техните преводи на Български
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Alcoholic and non-alcoholic steatohepatitis.
Estimates the steatohepatitis of the liverSteatoTest.
Alcoholic liver diseases(steatosis and steatohepatitis).
Estimates alcoholic steatohepatitis of the liverAshTest.
Steatohepatitis- is a phenomenon, which is observed in liver tissue, occurs with varying degrees of severity.
Estimates non-alcoholic steatohepatitis of the liverNashTest.
Over 95% of tests are interpretable and enable an effective diagnosis of fibrosis,steatosis and steatohepatitis.
Fatty liver may lead to inflammatory disease(i.e. steatohepatitis) and, eventually, cirrhosis.
Studies on rodents give promising results in the prophylaxis of alcoholic and nonalcoholic steatohepatitis.
Patients with results suggesting the presence of steatohepatitis or fibrosis should be considered for liver biopsy.
Milk thistle can help to treat the condition andstop it from becoming its more serious form, steatohepatitis.
Steatohepatitis is characterized by the accumulation of fatty material in the liver and is often associated with diabetes or obesity.
Alcoholic hepatitis has also a number of other titles,the most known from which is steatohepatitis or fatty liver.
The liver injury may vary from pure steatosis, steatohepatitis to fibrosis, cirrhosis and clinically overt liver failure.
Mipomersen treatment can cause liver enzyme elevations and hepatic steatosis andthis may induce steatohepatitis.
Regular screening for steatohepatitis/fibrosis should be performed at baseline and on an annual basis using the following imaging and biomarker evaluations.
From simple and uncomplicated fatty liver(steatosis),to the more serious fatty liver with inflammation(steatohepatitis or alcoholic hepatitis), to cirrhosis.
If a patient has biopsy-proven steatohepatitis or fibrosis, the benefit-risk should be reassessed and treatment stopped if necessary.
The purpose of the investigation was to estimate the risk of cirrhosis andliver cancer in people with nonalcoholic fatty liver disease(NAFLD) or steatohepatitis(NASH).
Once that happens, if excess alcohol is not involved,the condition is called nonalcoholic steatohepatitis(steato- for fat and- hepatitis because the liver is inflamed).
Clinicians should look for metabolic risk factors and alternate etiologies for hepatic steatosis in patients who haveother types of chronic liver disease and who also have steatosis and steatohepatitis.
The steatohepatitis score[or acute alcoholic hepatitis(NASH)] assesses the activity according to a consensual 3-grade diagnostic classification from a score combining steatosis, necrosis and inflammation(Kleiner NAS score).
Concern remains about the potential progression of fatty liver disease to steatohepatitis and fibrosis, for which monitoring of patients at risk of developing inflammatory and fibrotic changes includes repeated liver biopsy.
The former presents with liver steatosis and immunological constellation of higher IgA, ANA(1:320), ASMA(1:160),anti-SLA/LP positive antibodies and normal aminotransferases which leads to the diagnosis of AIH type 1 overlapped with steatohepatitis.
Fatty liver disease raises the risk of developing Type 2 diabetes and heart disease, andit can progress to a more severe condition called nonalcoholic steatohepatitis, or NASH, which is a leading cause of liver cancer, cirrhosis and liver transplants.
Researchers from the Department of Gastroenterology and Liver Diseases at the University of Kuala Lumpur Medical School in Malaysia conducted a randomized, double-blind,placebo-controlled trial to investigate the effectiveness of silymarin in patients with nonalcoholic steatohepatitis[NASH].
Although cases of hepatic dysfunction(elevated aminotransferase with increase in bilirubin or International Normalized Ratio[INR]) or hepatic failure have not been reported,there is concern that lomitapide could induce steatohepatitis, which can progress to cirrhosis over several years.
For the individual patient, the worst case scenario could be that they might not obtain the potential benefit of mipomersen in terms of reduced CVS morbidity/mortality because they cannot tolerate long-term treatment, butmight be harmed by progressive liver disease resulting from mipomersen-induced steatohepatitis.
As liver biopsy was not performed on a regular basis in the mipomersen study programme, it is not clear whether a small or a significant proportion of patients with mipomersen-induced steatosis also had inflammatory changes and fibrosis,i.e. might develop steatohepatitis, which may not be reversible after stopping the treatment.