Примери коришћења Endocannabinoids на Енглеском и њихови преводи на Српски
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But endocannabinoids travel in the opposite direction.
You can have too many receptors but not enough endocannabinoids.
These are known as endocannabinoids because they're produced within the body.
When a message passes from the one neuron to the next,the receiving neuron releases endocannabinoids.
Feedback from endocannabinoids slows down rates of neural signaling.
The third component of the endocannabinoid system is the enzymes that make the endocannabinoids and break them down.
Cannabinoid receptors and endocannabinoids can be found throughout the entire human body;
Raichlen says there are two leading theories on why running causes increased levels of endorphins and endocannabinoids.
These cannabinoids are known technically as endocannabinoids because they're made inside your body.
Like endocannabinoids, THC slows down signaling by binding to cannabinoid receptors.
These drugs, called endocannabinoid metabolic enzyme inhibitors,can increase the amount of endocannabinoids in the eye.
Endocannabinoids are substances produced from within the body that activate cannabinoid receptors.
GPR55 is activated by the plant cannabinoids Δ9-THC, and the endocannabinoids anandamide, 2-AG, noladin ether in the low nanomolar range.
Those endocannabinoids travel backward to influence the sending neuron- essentially giving it feedback from the receiving neuron.
But it binds to receptors all over this sprawling,diffuse system at once, whereas endocannabinoids are released in a specific place in response to a specific stimulus.
Endocannabinoids are molecules that, together with their receptors, can be found throughout the body- in our brains, organs, tissues, and even in our immune cells.
Since some studies have suggested that cannabis has cancer-fighting properties,the researchers wanted to see whether the naturally occurring endocannabinoids would have the same effect.
Endocannabinoids are important endogenous signaling molecules in our body and the endocannabinoid system is thought to be one of the body's natural defense systems against injury.
The chemical description includes an array of unique chemical classes: the non-classical cannabinoids, the classical cannabinoids associated with THC and CBD,the eicosanoids linked to the endocannabinoids.
Endocannabinoids such as 2-arachidonoyl glycerol(2-AG) and arachidonoyl ethanolamide are biologically active lipids that are involved in a number of synaptic processes including activation of cannabinoid receptors.
However, several other classes can technically be termed eicosanoid, including the eoxins, hepoxilins, resolvins, isofurans, isoprostanes, lipoxins, epi-lipoxins,epoxyeicosatrienoic acids(EETs) and endocannabinoids.
Local pharmacological andgenetic manipulations revealed that endocannabinoids and exogenous cannabinoids increased odor detection and food intake in fasted mice by decreasing excitatory drive from olfactory cortex areas to the MOB.”.
Stemming from the centuries-old and well known effects of Cannabis on intestinal motility and secretion, research on the role of the endocannabinoid system in gut function and dysfunction has received everincreasing attention since the discovery of the cannabinoid receptors and their endogenous ligands, the endocannabinoids.
Someone having decreased endocannabinoid count could come about because they have too few receptors or they too few endocannabinoids, but ultimately the body tries to keep these in balance so that the systems work at their best.
Synthetic cannabinoids encompass a variety of distinct chemical classes: the classical cannabinoids structurally related to THC, the nonclassical cannabinoids(cannabimimetics) including the aminoalkylindoles, 1,5-diarylpyrazoles, quinolines, andarylsulfonamides as well as eicosanoids related to endocannabinoids.
More technically, the authors of said study stated,“Local pharmacological andgenetic manipulations revealed that endocannabinoids and exogenous cannabinoids increased odor detection and food intake in fasted mice by decreasing excitatory drive from olfactory cortex areas to the MOB”.