Ví dụ về việc sử dụng Acinetobacter trong Tiếng anh và bản dịch của chúng sang Tiếng việt
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Serotyping of Acinetobacter calcoaceticus.
Acinetobacter poses very little risk to healthy people.
PER-1 is also common in multiresistant acinetobacter species in Korea and Turkey.
Acinetobacter infections rarely occur outside of healthcare settings.
The next slide I'mabout to show you is of carbapenem resistance in acinetobacter.
Acinetobacter baumannii accounts for about 80% of reported infections.
Diagnosed with pancreatitis, Patterson was flown for treatment to Frankfurt,where physicians discovered that he was infected with a deadly superbug called Acinetobacter baumannii.
Acinetobacter is often resistant to many commonly prescribed antibiotics.
In one test-tube study, acetic acid prevented the growth of bacteria,such as Acinetobacter baumannii, that commonly cause infections in burn patients.
Acinetobacter Infections: Alternative to imipenem or meropenem for treatment of infections caused by Acinetobacter.
However, it appears that LPS is nonessential in at least some Gram-negative bacteria, such as Neisseria meningitidis,Moraxella catarrhalis, and Acinetobacter baumannii.
Acinetobacter Baumannii- a common bacteria which is resistant to most antibiotics and which can easily infect patients in a hospital.
Because of its synergistic effect with beta-lactams, Arbekacin also holds promise as a treatment for multidrug-resistant Gram-negative bacterialinfections such as multidrug-resistant Pseudomonas aeruginosa and Acinetobacter baumannii.[4].
Acinetobacter causes a variety of diseases, ranging from pneumonia to serious blood or wound infections and the symptoms vary depending on the disease.
Scientists from the Research Group Experimental Ecology and Evolution at the Max Planck Institute for Chemical Ecology in Jena,Germany addressed this question using the soil bacterium Acinetobacter baylyi and the gut microbe Escherichia coli.
Acinetobacter baumanni is another pathogen that is likely to infect surgical wounds and it is found to be resistant to a variety of drugs.
Hospitalized patients, especially very ill patients on a ventilator, those with a prolonged hospital stay, those who have open wounds, or any person with invasive devices like urinarycatheters are also at greater risk for Acinetobacter infection.
Acinetobacter may also“colonize” or live in a patient without causing infection or symptoms, especially in tracheostomy sites or open wounds.
Colistin is not much used in people because it can damage their kidneys,but it is a vital last line of defence against Acinetobacter, Pseudomonas aeruginosa, Klebsiella and Enterobacter, two of which are specifically mentioned on the CDC watch list.
They include Acinetobacter, Pseudomonas, and various Enterobacteriaceae(including Klebsiella, E coli, Serratia, and Proteus), which can cause severe and often deadly infections, such as bloodstream infections and pneumonia.
A dose of up to 2 g three times daily in adults and adolescents and a dose of up to 40 mg/kg three times daily in children may be particularly appropriate when treating some types of infections, such as infections due to less susceptible bacterial species(e.g. Enterobacteriaceae,Pseudomonas aeruginosa, Acinetobacter spp.), or very severe infections.
The activity of sulbactam against Acinetobacter spp. seen in in-vitro studies makes it distinctive compared to other β-lactamase inhibitors, such as tazobactam and clavulanic acid.[3].
However, resistant pathogens may produce β-lactamase enzymes that can inactivate ampicillin through hydrolysis.[3] This is prevented by the addition of sulbactam, which binds and inhibits the β-lactamase enzymes.[2][3]It is also capable of binding to the PBP of Bacteroides fragilis and Acinetobacter spp., even when it is given alone.
There is a serious lack of treatment options for multidrug-and extensively drug-resistant M tuberculosis and gram-negative pathogens,including Acinetobacter and Enterobacteriaceae(such as Klebsiella and E. coli), which can cause severe and often deadly infections that pose a particular threat in hospitals and nursing homes.
Acinetobacter baumannii Stenotrophomonas maltophilia Haemophilus influenzae Moraxella catarrhalis Neisseria gonorrhoeae Enterobacteriaceae Escherichia coli(including ESBL-producing strains) Klebsiella pneumoniae(including carbapenem resistant strains) Klebsiella oxytoca Enterobacter species Citrobacter species Proteus mirabilis Serratia marcescens.
There is a serious lack of treatment options for multidrug-and extensively drug-resistant M tuberculosis and gram-negative pathogens,including Acinetobacter and Enterobacteriaceae(such as Klebsiella and E. coli), which can cause severe and often deadly infections that pose a particular threat in hospitals and nursing homes.
Plazomicin has been reported to demonstrate in vitro synergistic activity when combined with daptomycin or ceftobiprole versus methicillin-resistant Staphylococcus aureus(MRSA), vancomycin-resistant S. aureus(VRSA) and against Pseudomonas aeruginosa when combined with cefepime, doripenem, imipenem or piperacillin/tazobactam.[1]It also demonstrates potent in vitro activity versus carbapenem-resistant Acinetobacter baumannii.[3].
It was developed in response to the growing rate of antibioticresistant bacteria such as Staphylococcus aureus, Acinetobacter baumannii, and E. coli.[2] As a tetracycline derivative antibiotic, its structural modifications has expanded its therapeutic activity to include Gram-positive and Gram-negative organisms, including those of multi-drug resistance.
Among the priority pathogens is a drug-resistant tuberculosis, which kills about 250,000 people around the world each year,and a variety of multidrug resistant strains- Acinetobacter, Pseudomonas and various Enterobacteriaceae- which are responsible for infections in hospitals and nursing homes and among patients whose care requires ventilators and catheters.