Ví dụ về việc sử dụng Vegf trong Tiếng anh và bản dịch của chúng sang Tiếng việt
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VEGF had high sensitivity and specificity.
Bevacizumab targets circulating VEGF ligand.
VEGF can be blocked with certain drugs.
YZ-III 3ml* 0.1u increments VEGF delivery device.
VEGF levels correlate with disease activity.
Cancers that can express VEGF are able to grow and metastasize.
VEGF is important for the growth of new blood vessels.
One of the proteins responsible for this process is known as VEGF.
VEGF and other growth factors are involved in cancer angiogenesis.
Ramucirumab blocks the effect of the protein VEGF and may prevent the growth of new blood vessels that tumors need to grow.
VEGF165 appears to be the major gene product found in human tissue andis the most effective angiogenic factor in the VEGF family.
Scientists assessed the whole genome of the animals andfound the CRISPR-Cas9 complex modified only the VEGF gene and did not affect other genes.
The normal function of vegf is to create new blood vessels during embryonic development, after an injury or to bypass blocked blood vessels.
A previous study by these authorsdemonstrated that low-energy ESWT promoted expression of VEGF in damaged neural tissue and improved locomotor function after spinal cord injury(SCI).
This medication blocks the VEGF, a protein that stimulates blood vessel growth, and prevents the growth of new abnormal blood vessels in the retina.
All these drugs are kinase inhibitors that interfere with multiple tyrosine kinase pathways,including VEGF and RET, that play a role in cancer cell growth and formation of blood vessels that supply tumors.
Six forms of human VEGF mRNA encoding VEGF proteins of 121, 145, 165, 183, 189, and 206 amino acids are produced from a single gene as a result of alternative splicing.
When researchers at the National Cancer Institute gave FBL 101 to mice with prostate cancer, they found that it decreased atumor blood-vessel growth factor called VEGF to undetectable levels.
The results showed that only PD98059 blocks VEGF expression, suggesting an important role for ERK1/2 in regulating VEGF expression in A549 cells.
For example, HA hydrogels are appealing for engineering vasculature from endothelial progenitor cells byusing appropriate growth factors such as VEGF and Ang-1 to promote proliferation and vascular network formation.
In AMD, VEGF causes the formation of new blood vessels in the eyes but also leads to leakages of blood and fluid into the eye, damaging an area at the center of the retina called macula.
A quinoline urea derivative, tivozanib suppresses angiogenesis by being selectively inhibitory against vascular endothelial growth factor.[4] It was developed by AVEO Pharmaceuticals.[5]It is designed to inhibit all three VEGF receptors.
When VEGF and other endothelial growth factors bind to their receptors on endothelial cells, signals within these cells are initiated that promote the growth and survival of new blood vessels.
April 2014- One in five people over age 75 suffer from age-related macular degeneration(AMD), and currently the only effective treatment for the advanced"wet" form ofAMD involves injections of medicines called VEGF inhibitors into the eye.
Injections of anti-VEGF drugs are the most common treatment against AMD,but at least seven injections per year are required, because VEGF is continuously over-expressed by the cells of the diseased retinal pigment epithelium.
Furthermore, these factors attach to receptors including the VEGF receptor VEGF-R2 and the Angiopoietin receptor Tie-2, both expressed upon a substantial percentage of progenitor cells, further enhancing tissue function and effectiveness.
When VEGF and other endothelial growth factors bind to their receptors on endothelial cells, signals within these cells are initiated that promote the growth and survival of new blood vessels, which are necessary for tumor growth.
Ciglitazone significantly decreases VEGF production by human granulosa cells in an in vitro study, and may potentially be used in ovarian hyperstimulation syndrome.[5] Ciglitazone is a potent and selective PPARγ ligand.
Pegaptanib specifically binds to the 165 isoform of VEGF, a protein that plays a critical role in angiogenesis(the formation of new blood vessels) and increased permeability(leakage from blood vessels), two of the primary pathological processes responsible for the vision loss associated with neovascular AMD.