Exemplos de uso de Cellular rejection em Inglês e suas traduções para o Português
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Pulsed Doppler and diagnosis of cellular rejection> 3A.
Cellular rejection usually responds properly to pulse corticosteroid therapy.
Tissue Doppler and diagnosis of cellular rejection> 3A.
The acute cellular rejection occurs in 50-75% of patients, most commonly in the first 90 days.
The gold standard for the diagnosis of acute cellular rejection is histology.
Acute cellular rejection has been a common cause of graft loss and an indication for re-transplantation.
Transbronchial biopsy TBB is the primary method for diagnosing acute cellular rejection.
The diagnosis of acute cellular rejection is performed by clinical, endoscopic and pathologic anatomy.
Higher degree of fractal dimension were directly associated with progressively larger myocardial cellular rejection.
Most episodes of cellular rejection are asymptomatic in its early stage or may have nonspecific symptoms.
This was confused with other conditions that lead to ventricular dysfunction with cellular rejection and pulmonary thromboembolism.
Therefore, it differs from early acute cellular rejection, which occurs less than three months after liver transplantation.
In addition, it has been used to assess the left ventricle of patients submitted to cardiac transplantation,contributing to quantify myocardial cellular rejection.
Patients with steroid-resistant cellular rejection were treated with mycophenolate mofetil and anti-T-cell monoclonal antibody.
Introduction: this study aims to correlate the number of hla mismatches with the frequency of acute cellular rejection in the first six months after heart transplantation.
However, cellular rejection is frequently found in those patients- approximately 70% of them have that complication in the first year after CT.
Doses greater than 0.15 mg/kg/day were associated with the prevention of acute cellular rejection but predisposed patients to infectious disease.
Acute cellular rejection is frequent during the 1st posttransplant year incidence of up to 65% and normally does not represent an immediate risk to the patient.
It was found that the calculation of fractal dimension can contribute to the assessment of myocardial cellular rejection in patients undergoing endomyocardial biopsy after cardiac transplantation.
Methods: retrospective study, single center study that evaluated 226 patients who received a kidney graft between 2008 and 2013 andwhere treated with atg either for induction therapy or for treatment a severe acute cellular rejection.
Studies have reported that episodes of cellular rejection do not increase the risk of cardiovascular death including myocardial infarction, arrhythmias, sudden death and CAV.
Additionally, the high hydrophillic property of the glucosaminoglycans present in the valve leaflet leads to increased oncotic pressure in the extracellular matrix during cellular rejection, thus causing edema and precluding adequate function.
Despite advances in immunosuppressive therapy over the past decade, acute cellular rejection CR of the donor heart remains an important factor related to long-term morbidity and mortality.
The acute cellular rejection is the leading cause of morbidity and mortality between 30 days and 12 months post-transplant, along with chronic rejection and malignancies, is also a major cause of death in the late post-transplant.
Despite advances in immunosuppressive therapy in the last decade, the acute cellular rejection of the transplanted heart remains an important factor associated with longterm morbidity and mortality 22.
The objective of the present study was to determine whether acute cellular rejection correlates with lactate dehydrogenase LDH levels, proinflammatory cytokine levels, and differential cell counts in the pleural fluid of lung transplant recipients.
Our results showed the feasibility of TDI for early diagnosis of significant cellular rejection and that TDI added diagnostic information concerning conventional echocardiogram for such issue.
By means of endomyocardial biopsies, foci of grade II andIII acute cellular rejection were found in our patient, and were controlled with the initiation of a corticosteroid until signs of rejection could no longer be observed.
Recent studies have suggested that the direct activation of the recipient's immune system can induce cellular rejection episodes, and that the episodes of chronic rejection and CAV are more often associated with indirect activation of the immune system.
Hospital readmission of these patients is more frequent in comparison with other transplants and is generally associated with infection, rejection, dehydration, andgastrointestinal complications. Acute cellular rejection still shows a high frequency when compared to that of transplants of other solid organs, occurring in 50 to 75% of patients, most commonly during the first trimester, having a direct impact on the long-range result of the graft.