Exemplos de uso de Cerebral calcifications em Inglês e suas traduções para o Português
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In the studyby Vasconcelos-Santos et al., the prevalence of cerebral calcifications was 20.5%; neuroimaging studies included radiographs and ultrasound.
Therefore, cerebral calcifications were seen in 35 patients 53.8%; 95% CI: 41.6% to 65.6% and retinochoroiditis was detected in 30 46.1%; 95% CI: 34.3% to 58.3% within the first year of life.
The clinical picture of toxoplamosis mainly shows hydrocephalus or microcephaly, diffuse cerebral calcifications in cerebral cortex, microphthalmia and chorioretinitis due to microcephaly.
Cerebral calcifications and retinochoroiditis were detected in 25 patients 38.4%, ten 15.3% had only calcifications, four had only retinochoroiditis, and one patient had retinochoroiditis and did not undergo imaging examination; for statistical purposes, this patient was added to those that had only retinochoroiditis, totaling five cases 7.7% in this category Fig.
Additionally, they concluded that,amongst the techniques for diagnosis of cerebral calcifications by MRI, SWI was the most frequently indicated modality to detect calcifications in oligodendrogliomas with results confirmed by CT image.
The studied variables were the time of screening, presence of Toxo-IgM in the first month of life, age when the Toxo-IgM became negative, gestational age, maternal and infant treatment,presence of cerebral calcifications, and presence of retinochoroiditis within the first year of life.
The imaging findings in that cohort consisted of cerebral calcifications, predominantly periventricular, but also of calcifications in the brain parenchyma, thalamus, and basal ganglia.
There are also case reports that used other imaging methods, such as obstetric ultrasound to carry out intrauterine investigation of pregnant women with a history of ZikaV infection,which showed gross cerebral calcifications, abnormalities in the cerebellar vermis and corpus callosum dysgenesis.
Other studies that also detected a lower prevalence of cerebral calcifications than that of the present study generally included skull radiographs among the neuroimaging tests, which have known low sensitivity to detect calcifications. .
They are indicated for patients with neurological abnormalities of indefinite diagnoses,particularly for difficult to control ataxia and epilepsy associated with cerebral calcifications. They are indicated for patients with osteopenia/osteoporosis and sufferers from dermatitis herpetiformis.
The prevalence of clinical manifestations in this sample of patients more than 60% with retinochoroiditis and/or cerebral calcifications appears to confirm the higher morbidity of the infection in Brazilian and South American children in general, when compared to children from other continents, such as Europe and North America, where clinical manifestations are described at birth in approximately 40% of infected newborns.
Among the 65 patients included, 40 61.5%; 95% confidence interval ENT[CIENT]: 49.3% to 72.7% had some clinical alteration within the first year of life,considering only retinochoroiditis and cerebral calcifications without including other central nervous system abnormalities, such as ventricular dilation.
Untreated CD often manifests monosymptomatically, as refractive anemia, dermatitis herpetiformis, which can be considered as the dermatolological expression of the disease, delayed menarche and early menopause, infertility, repeated miscarriages, hypertransaminemia, depression, progressive neurological symptomology, primarily ataxia andepilepsy associated with cerebral calcifications, osteoporosis and hypoplasia of dental enamel.
The ipsilateral leptomeninge angiomatosis also represents one of the main signs of the syndrome, may present with a progressive nature andcommonly leads to cerebral calcifications, epileptic convulsive crises, contralateral hypertrophy and hemiparesis and mental retardation 10.
The main objective of the imaging assessment of a possible intrauterine infection by ZikaV is the detection of neurological complications related to transplacental infection,such as microcephaly, cerebral calcifications, neuronal migration disorders, loss of brain tissue, and ventricular system dilation.
Congenital infection by cytomegalovirus can occur at any stage of pregnancy or during childbirth. The teratogenic potential is proportional to the early infection.The systemic manifestations in which there are cerebral calcifications, hepatosplenomegaly, chorioretinitis, thrombocytopenia, macular rash, hemolytic anemia and organ development impaired is called cytomegalic inclusion disease.
Although SWI is already a recognized technique,computed tomography CT is still considered to be a conclusive technique in the differential diagnosis of cerebral calcification.
Despite this, some individuals develop these calcium deposits and have no well-defined etiology,cases in which the cerebral calcification is characterized as Fahr's disease.
The majority of cases presented an imaging pattern that suggested congenital infection- cerebral calcification, ventriculomegaly and lissencephaly- and their mothers presented clinical conditions that suggested infection by Zika virus, with rash and pruritus during the months of fetus development.
Infected children may present, at birth, signs and symptoms, such as hydrocephalia,chorioretinitis, cerebral calcification, and mental retardation, or they may be born normal and develop sequelae during childhood or even in adulthood.