Exemplos de uso de Inflammatory cascade em Inglês e suas traduções para o Português
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Once bound to the specific particle,the TIR activates its inflammatory cascade.
The activity of serotonergic inflammatory cascade has been speculated to provoke the FMF attacks.
Analogous to the apoptosome, which activates apoptotic cascades, the inflammasome activates an inflammatory cascade.
MSU crystals trigger the inflammatory cascade, which ultimately result in pain and inflammation.
These drugs were developed to reach the specific goals in the inflammatory cascade that leads to the disease.
There is disruption of the cell membrane, with leakage from the cytosol to the extracellular medium,leukocyte margination and activation of the inflammatory cascade.
Knowledge of pa involved in the inflammatory cascade is important to the search for markers of disease severity 3.
Additionally, it was shown that these abnormalities were partially mediated by TNF-alfa a known mediator of the inflammatory cascade.
The tumoral necrosis factor? TNF-?is a key cytokine in the inflammatory cascade, and one of the main mediators for sepsis and ALI/ARDS.
Corticosteroids are drugs with potent anti-inflammatory and even immunosuppressive action,acting at an early stage of the inflammatory cascade.
Local anesthetics have shown to be effective in modulating the inflammatory cascade during ischemia and reperfusion of the heart, lungs, and liver.
It would not affect symptoms, butthe entire allergic process by impacting the development of the allergic inflammatory cascade.
They also contribute to perpetuate cytokines and chemokines inflammatory cascade, as well as to stimulate histamin release by mast cells.
In particular:¤ Some common lectins, such as gliadin, present in wheat gluten(and also in other cereals), act to trigger the inflammatory cascade.
Mutant pyrin is associated with uncontrolled inflammatory cascade, probably by dysregulated inflammasome function and excessive IL-1? production?
Biological therapy comprises a group of drugs designed to block specific stages of the inflammatory cascade implied in psoriasis pathogenesis.
The inflammatory cascade activation occurs, followed by coagulation activation, hemolysis and thromboembolytic phenomena are generated, all contributing to multiple-organ injury.
The deposition of circulating immune complexes in perivascular dermis triggers the inflammatory cascade, which causes vascular injury and consequent fibrosis.
The anti-inflammatory effect may also be related to decreased graft rejection via its immunomodulatory effects andthe inhibition of factors that activate the inflammatory cascade.
The accumulation of abnormal proteins in the endoplasmic reticulum activates the inflammatory cascade, favors apoptosis and interferes with normal protein synthesis.
With the onset of surgical trauma and inflammatory cascade activation that happens even after the surgery, there is the release of inflammatory mediators and its corresponding nociceptors sensitization.
The pathogenesis of the organic dysfunction which occurs after extracorporeal circulation involves the inflammatory cascade and cellular components of the immune system.
Restoration of the blood supply after an ischemic period activates an inflammatory cascade associated with multiple processes, including ion accumulation, free reactive oxygen species(ros) formation, endothelial dysfunction, and immune activation.
Experimental models of surfactant deficiency showed that low-volume MV induces cytokine release and initiates the inflammatory cascade, which also occurs in volutrauma.
Activation of the mature dendritic cells myeloid andplasmacytoid initiates the inflammatory cascade, with differentiation of the lymphocytes in the Th1 and Tc1 lineages by costimulation, consisting of the interaction of non antigen-specific cells.
A number of different cyclooxygenase inhibitors indomethacin,ibuprofen appear to have beneficial effects at specific points in the inflammatory cascade and on the survival of animals.
One of those elements is TNF-?,cytokine capable of participating in the inflammatory cascade activating both epidermal keratinocytes and endothelial cells and synoviocytes.
Such inflammatory cascade may contribute to the development of postoperative complications, including respiratory failure, renal dysfunction, hemorrhagic disorders, neurological dysfunction, changes in liver function, and finally failure of multiple organs.
These enzymes are involved in coagulation,fibrinolysis, and in the inflammatory cascade, demonstrating the capacity of aprotinin to modulate several systemic processes.
The fluid overload in ckd patients was associated to the presence of elevated serum levels of endotoxins,which leads to activation of inflammatory cascade contributing to maintaining a constant inflammatory state observed in this population.