Exemplos de uso de Node dysfunction em Inglês e suas traduções para o Português
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Sinus node dysfunction is treated with a pacemaker.
They can be classified broadly into SINOATRIAL NODE dysfunction and ATRIOVENTRICULAR BLOCK.
Sinus node dysfunction may cause episodes of sino-atrial block with bradycardia or ectopic atrial tachycardia.
Examples include sinus node dysfunction and heart block.
Pacemaker is indicated in presence of documented sinus node disease related to symptomatic bradycardia or symptomatic chronotropic dysfunction, andin cases the drug causing node dysfunction cannot be interrupted or modified.
YES NO Does the patient have severe bradycardia, sinus node dysfunction or second and third degree heart block, in the absence of a pacemaker?
This mutation may increase glucose uptake by stimulating translocation of the glucose transporter GLUT-4 to the plasma membrane and increase hexokinase activity,thus leading to glycogen accumulation, which may cause sinus node and atrioventricular node dysfunction.
Second or third degree atrioventricular(AV)block or sinus node dysfunction, unless these patients have a functioning artificial pacemaker.
Sinus node dysfunction after Cox-Maze surgery was considered temporary[19], because studies on patients operated for chronic atrial fibrillation secondary to organic lesion demonstrated dysfunction in 12 of 15 cases at 3 postoperative months, with progressive improvement until the patient reached 12 postoperative months.
In addition, the cardiac excito-conduction tissue is frequently damaged,leading to sinus node dysfunction and disorders of the atrioventricular and intraventricular conduction.
Atrial fibrillation or sinus node dysfunction patients, especially those with bradycardia and tachycardia alternation, show high risks for thromboembolic occurrences, even after pacemaker placement.
Benchimol et al. used EPS to diagnose a case of permanent atrial standstill and the presence of sinus node dysfunction in patients with chronic Chagas heart disease.
Another important clinical component of sinus node dysfunction was the syndrome bradycardia-tachycardia, which was diagnosed with the EPS for the first time by Pimenta et al. in a patient with chronic Chagas heart disease.
Arrhythmias may be explained by reentrant mechanisms caused by suture lines in the atrium,whereas sinus node dysfunction may occur due to direct injury of the node or its artery.
In this report, we describe an unusual case of a toddler presenting with sinus node dysfunction, flutter and atrial fibrillation, atrioventricular block, prolonged HV interval and family history of sudden death, probably due to mutations in the SCN5A gene, which, in this case, was characterized as a compound heterozygote p. G400R and p. T1461S.
The patient reported was a child diagnosed with left atrial isomerism associated with a sinus venosus atrial septal defect and sinus node dysfunction with sinus bradycardia, sinus pauses and junctional rhythm.
Dynamic Electrocardiogram- Twenty-four-hour EKG recording confirmed sinus node dysfunction diagnosis by showing bradycardia sinus rhythm alternating with atrial ectopic episodes and bradycardia stress periods and up to 4.3 sec pauses, especially between midday and 1pm, 2.20 pm and 3.30 pm, and 8.45 pm and 9.05 pm.
In diabetic patients, the increased glycogen concentration in the ventricles is apparently associated with the severity of diabetes, and makes the heart more susceptible to the effects of ischemia,in addition to leading to cardiac malfunctioning, since its accumulation in the conductive tissue is the cause of sinus and atrioventricular node dysfunction.
In two patients with PLE after the Fontan operation and associated sinus node dysfunction, the implantation of a pacemaker increased cardiac output and resolved the syndrome.
Sinus node disease comprises any node dysfunction and unsuitable sinus bradycardia characterized by heart rate lower than 60 bpm, which does not properly increase with exercise. Sinus pause longer than 3 seconds, sinus-atrial block, and brady-tachyarrhythmia defined by sinus or junctional bradycardia, alternating with atrial tachycardia or paroxystic atrial fibrillation.
This highlighted the importance of the potential participation of both sinus node dysfunction and AV nodal dysfunction in the genesis of arrhythmias in patients with this condition.
The procedure proposed by the authors, posteriorly named lateral tunnel, which consisted of to create a tunnel between the holes in the inferior and superior vena cava with a concuit prosthesis sutured on the lateral side of the right atrium provided a more laminar flow system, but it did not resulte in a significant reduction in the incidence of complications,particularly atrial arrhythmias and sinus node dysfunction 6,7.
All studies excluded patients with permanent atrial fibrillation AF, sinoatrial SA node dysfunction, atrioventricular blockage or any other indication for the implantation of a permanent pacemaker.
Some late complications may be observed, many related to technical aspects of the surgical correction such as obstruction of the superior vena cava in 10% of the cases, obstruction of the inferior vena cava in 2%, interatrial leaks, obstruction of the pulmonary veins,atrial arrhythmias sinus node dysfunction, right ventricular dysfunction, and tricuspid insufficiency, this last probably due to annular dilatation as a consequence of right ventricular dysfunction. .
These findings are consistent with the literature,which shows a high incidence of sinus node dysfunction and low total atrioventricular block, as well as significant atrial and ventricular arrhythmias are uncommon 18.
It is noteworthy that the IgGs of patients who presented ventricular arrhythmias increased the frequency of heart rate and the cAMP production 75%,whereas the IgGs of patients with sinus node dysfunction decreased the frequency of heart rate and increased the synthesis of phosphatydilinositol 76.9%, in rat cardiomyocyte culture.
Second- or third- degree atrio-ventricular block, complete bundle branch block, distal block,sinus node dysfunction, atrial conduction defects, or sick sinus syndrome except when used in conjunction with a functioning pacemaker.
Patients with prolonged QT at baseline(uncorrected> 440 msec), or severe bradycardia,sinus node dysfunction or second degree and third degree heart block in the absence of a pacemaker.