Exemple de utilizare a Prostaglandin synthesis în Engleză și traducerile lor în Română
{-}
-
Medicine
-
Colloquial
-
Official
-
Ecclesiastic
-
Ecclesiastic
-
Computer
-
Programming
Meloxicam acts by inhibition of prostaglandin synthesis.
Suppression of prostaglandin synthesis is negativeaffects the intrauterine development of the fetus.
Meloxicam acts by inhibition of prostaglandin synthesis.
Inhibition of prostaglandin synthesis may adversely affect the pregnancy and/or the embryo/foetal development.
The(+) enantiomer is more potent in inhibiting prostaglandin synthesis.
However, inhibition of prostaglandin synthesis might adversely affect pregnancy.
These effects are expected following inhibition of prostaglandin synthesis.
These products act by hindering the prostaglandin synthesis, substances that cause inflammation and pain.
Firocoxib is a non-steroidal anti-inflammatory drug(NSAID)that acts by selective inhibition of cyclooxygenase-2(COX-2) mediated prostaglandin synthesis.
Cardiovascular, have been reported in animals given a prostaglandin synthesis inhibitor during the organogenetic.
Firocoxib is a non-steroidal anti-inflammatory drug(NSAID) belonging to the Coxib group,which acts by selective inhibition of cyclooxygenase-2(COX-2) mediated prostaglandin synthesis.
From the beginning of the sixth month of pregnancy, all prostaglandin synthesis inhibitors may expose.
In animals, administration of a prostaglandin synthesis inhibitor has been shown to result in reproductive toxicity(see section 5.3).
Data from epidemiological studies suggest an increased risk of miscarriage after use of prostaglandin synthesis inhibitors in early pregnancy.
In animals, administration of a prostaglandin synthesis inhibitor has been shown to result in increased pre- and post-implantation loss and embryo-foetal lethality.
Preclinical data also suggest that tissue ischaemia, allopurinol,calcium channel blockers and some prostaglandin synthesis inhibitors could interfere with PhotoBarr PDT.
Ketorolac, by inhibiting prostaglandin synthesis secondary to ocular surgical insult or direct mechanical stimulation of the iris, may also contribute to the prevention of surgically induced miosis.
In addition, increased incidences of various malformations,including cardiovascular, have been reported in animals given a prostaglandin synthesis inhibitor during the organogenetic period.
As with other medicinal products known to inhibit prostaglandin synthesis, fluid retention and oedema have been observed in patients taking celecoxib.
Bromfenac is a non-steroidal anti-inflammatory drug(NSAID)that has anti-inflammatory activity which is thought to be due to its ability to block prostaglandin synthesis by inhibiting primarily cyclooxygenase 2(COX-2).
Celecoxib, as with other medicinal products inhibiting prostaglandin synthesis, may cause uterine inertia and premature closure of the ductus arteriosus during the last trimester.
It plays a fundamental role in numerous metabolic and biochemical reactions such as DNA synthesis and repair,protein synthesis, prostaglandin synthesis, amino acid transport and enzyme activation.
It is rich in ursolic acid and silymarin,inhibiting prostaglandin synthesis involved in inflammatory mechanisms in the epidermis and protecting the membrane of living cells from irritating mechanical factors(such as improper use of scrub products).
Parecoxib sodium is suspected to cause serious birth defects when administered during the last trimester of pregnancy because as with other medicinal products known to inhibit prostaglandin synthesis, it may cause premature closure of the ductus arteriosus or uterine inertia(see sections 4.3, 5.1 and 5.3).
Since prostaglandin synthesis inhibition may result in deterioration of renal function and fluid retention, caution should be observed when administering Dynastat in patients with impaired renal function(see section 4.2) or hypertension, or in patients with compromised cardiac or hepatic function or other conditions predisposing to fluid retention.
Meloxicam is a NSAID of the oxicam class which acts by inhibition of prostaglandin synthesis, thereby exerting anti-inflammatory, anti-exudative, analgesic and antipyretic effects.
Selenium also has a role in prostaglandin synthesis by protecting the oxidative state of lipid intermediates formed during cyclooxygenase reactions which determines the balance of the end products and whether proaggregatory, pro-inflammatory or anti-aggregatory, anti-inflammatory responses will dominate.
Meloxicam is a non-steroidal anti-inflammatory drug(NSAID)of the oxicam class which acts by inhibition of prostaglandin synthesis, thereby exerting anti-inflammatory, analgesic, anti-exudative and antipyretic effects.
Selenium also has a role in prostaglandin synthesis by protecting the oxidative state of lipid intermediates formed during cyclooxygenase reactions which determines the balance of the end products and whether proaggregatory, pro-inflammatory or anti-aggregatory, anti-inflammatory responses will dominate.
Meloxicam is a non-steroidal anti-inflammatory drug(NSAID)of the oxicam class which acts by inhibition of prostaglandin synthesis, thereby exerting anti-inflammatory, analgesic, anti-exudative and antipyretic effects.