Examples of using The complement system in English and their translations into Vietnamese
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(3) Blood proteins, including members of the complement system and other mediators of inflammation.
These mechanisms include phagocytosis,antimicrobial peptides called defensins, and the complement system.
The complement system is made up of proteins that aid-- or complement-- antibodies that fight infections.
These mechanisms include phagocytosis,antimicrobial peptides called defensins, and the complement system.
The complement system is a set of circulating blood proteins that work together to promote immune and inflammatory responses.
Sometimes antibodies aregenerated against tumor cells allowing for their destruction by the complement system.
Gingivalis subverts the complement system and handicaps immune cells, rendering them less able to clear infection from the gum tissue.
It is a single chain variable fragment of amonoclonal antibody targeted against component 5 of the complement system.[4].
If there are defects in their structure, the complement system works either spontaneously or in response to thermal, chemical and cold stimuli.
It increases the production of prostaglandin E2 and reduces the production of leukotriene B4 in human white blood cells,and inhibits the complement system.
The complement system is made up of about 25 proteins that work together to“complement” the action of antibodies in destroying bacteria.
C3 and C4 are used to determine whether deficiencies orabnormalities in the complement system are causing, or contributing to, a patient's disease or condition.
Pseudomallei triggers both the complement system and coagulation cascade, however the thick bacterial capsule prevents deposition of the complement membrane attack complex.
It may also be used when a doctor suspects that someone may have an immune complex-related condition and he orshe wants to check the status of the complement system.
Activation of the complement system involves a sequential enzyme cascade in which the proenzyme product of one step becomes the enzyme catalyst of the next step.
This finding suggested to the scientists that the Toll-like receptorsignaling was somehow involved in“crosstalk” with the complement system, serving to augment the inflammatory response.
C1q is a protein complex and is the first component of the complement system, part of the innate immune system that improvesthe activity of phagocytes and antibodies.
Its physiological role is to bind to lysophosphatidylcholine expressed on the surface of dead or dying cells(and some types of bacteria)in order to activate the complement system via C1q.
This receptor is part of the complement system which is a component of the immune system which helps clear infection but can result in detrimental inflammation if incorrectly controlled.
Its physiological role is to bind to phosphatidylcholine expressed on the surface of dead or dying cells(and some bacteria)in order to activate the complement system via the C1Q complex.
Mannose-binding lectin(MBL) is a protein capable of activating the complement system, part of the innate immune system that improvesthe activity of phagocytes and antibodies.
Obinutuzumab binds to CD20 on B cells and causes these cells to be destroyed by engaging the adaptive immune system, directly activating intracellular apoptosis pathways,and activating the complement system.[1].
Meanwhile, other studies by the Penn group and others have shown that Toll-like receptors, or TLRs- a set of proteins that also activate immune cell responses-may act in concert with the complement system.
In the case of pseudo-allergic angioedema,the cause of the pathology is congenital defects of the complement system- a group of proteins that take part in the formation of primary immune and allergic reactions.
In the case of the pseudoallergic edema of Quincke,the cause of the pathology is the congenital defects of the complement system, a group of proteins that take part in the formation of primary immune and allergic reactions.