Voorbeelden van het gebruik van Nephrotoxicity in het Engels en hun vertalingen in het Nederlands
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Programming
Prevention of nephrotoxicity.
Nephrotoxicity is a dose-limiting side effect.
Female rats were found to be more susceptible to this nephrotoxicity.
Nephrotoxicity(kidney damage) is a major concern.
Increased ciclosporin levels have been associated with nephrotoxicity.
Nephrotoxicity of NSAIDs in dehydrated patients.
In addition, ataluren was found to increase nephrotoxicity of intravenous aminoglycosides.
Nephrotoxicity has been reported with the use of parenteral aminoglycosides.
ototoxicity or nephrotoxicity.
Nephrotoxicity was not observed during TOBI Podhaler clinical studies.
sensitive indicator of cidofovir-induced nephrotoxicity.
Nephrotoxicity has been associated with parenteral aminoglycoside therapy.
Severely maternally toxic doses to female rabbits(i.e. nephrotoxicity) lead to spontaneous abortions and death.
Increases the nephrotoxicity of cyclosporine and the toxicity of methotrexate;
The substance also enhances the harmful properties of cisplatin drugs and cephalosporins nephrotoxicity.
The signs of nephrotoxicity were partially reversible in some patients.
From cephalosporin antibiotics when combined with"torasemide" in a dose of 50 mg/ day appears nephrotoxicity property;
Azotemia is a type of nephrotoxicity that involves excess nitrogen compounds in the blood.
The likelihood of side effects of glucocorticosteroid drugs increases and the nephrotoxicity of cyclosporine increases.
There was no evidence of nephrotoxicity during clinical trials with inhaled tobramycin and Vantobra.
ototoxicity and nephrotoxicity see section 4.4.
Enhanced nephrotoxicity has been observed following the administration of amphotericin B and ibuprofen in conjunction with tacrolimus.
notable effects are the nephrotoxicity and the kidney tumours Dirheimer and Creppy, 1991.
Enhanced nephrotoxicity has been observed following the administration of amphotericin B and ibuprofen in conjunction with tacrolimus.
Preclinical animal studies demonstrated that nephrotoxicity was the major dose-limiting toxicity of cidofovir.
auditory functions(increasing oto- and nephrotoxicity of aminoglycosides);
Platinum compounds(risk of increased nephrotoxicity and ototoxicity); anticholinesterases,
Nephrotoxicity Dose-dependent nephrotoxicity is the major dose-limiting toxicity related to administration of cidofovir.
There is also a strong need for suitable pre-clinical in vitro kidney models to predict nephrotoxicity and study disease in the biopharmaceutical industry.
Dose-dependent nephrotoxicity is the major dose-limiting toxicity related to administration of cidofovir see section 4.8.