Приклади вживання Angiotensin Англійська мовою та їх переклад на Українською
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Angiotensin II.
Those are two cell types for angiotensin II to act on.
So angiotensin II is formed.
They're also referring to angiotensin II and all of its targets.
Angiotensin II will go out to a number of different places.
C09 Agents acting on the renin- angiotensin system.
Angiotensinogen becomes angiotensin I after meeting renin.
It should also take into account the treatment of angiotensin II.
The first acts as a blocker of angiotensin II, the second- as a diuretic.
We have talked about renin, we have talked about angiotensin.
Angiotensin II has both direct and indirect effects on blood pressure.
Among them there are antagonists of the angiotensin enzyme receptors.
And the angiotensin hormone actually gets that smooth muscle to constrict.
Antihypertensives: agents acting on the renin- angiotensin system(C09).
And just like the angiotensin 2, aldosterone is going to cause salt reabsorption.
So very quickly theblood vessels will start constricting if angiotensin 2 is around.
And aldosterone and angiotensin work in areas of the nephron that are permeable to water.
And so here's a little kidney here,and this kidney is going to be affected by angiotensin 2 very slowly by comparison.
Angiotensin I now floats through blood vessels and of course blood vessels have cells lining them.
Status of newborns whose mothers have taken angiotensin II receptor antagonists, should be carefully monitored for the presence of hypotension see.
Angiotensin II binds to specific receptors located on cell membranes in various tissues.
The functional role of these receptors is unknown,as well as the effect of their possible excessive stimulation with angiotensin II, the level of which increases telmisartan.
So we have talked about angiotensin 2, and we know that angiotensin 2 is a pretty small hormone.
The risk of developing the disease increases significantly with mutations in the genes,which are directly responsible for the synthesis of proteins of sodium channels, angiotensin and renin.
So if angiotensin II comes around, that would be one of the triggers to let the cholesterol turn into aldosterone.
For example, both ACTH, which stimulates cortisol secre-tion, and angiotensin II, which stimulates aldosterone secretion, increase the conversion of cholesterol to pregnenolone.
Angiotensin II receptor antagonists- in their action are similar to an ACE inhibitor, but have fewer side effects.
The main physiological effects of angiotensin II are vasoconstriction, stimulation of aldosterone production, regulation of the water-electrolyte state and stimulation of cell growth.
ACEi- angiotensin-converting enzyme inhibitors, ARB- inhibitors of angiotensin 2, CCB- calcium channel blockers, TD- thiazide diuretics.