Примери за използване на Active tubular на Английски и техните преводи на Български
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Active tubular secretion contributes to the renal elimination of glycopyrronium.
The renal clearance occurs through glomerular filtration and active tubular secretion.
This indicates that active tubular secretion is an important part of the elimination of tenofovir.
The renal clearance occurs through glomerular filtration and active tubular secretion.
This indicates that active tubular secretion is an important part of the elimination of tenofovir.
Emtricitabine is primarily excreted via glomerular filtration and active tubular secretion.
Tenofovir is primarily excreted by the kidney by both filtration and an active tubular transport system with approximately 70-80% of the dose excreted unchanged in urine following intravenous administration.
Tenofovir is renally eliminated by both glomerular filtration and active tubular secretion.
Co-administration of emtricitabine with medicinal products that are eliminated by active tubular secretion may increase concentrations of emtricitabine, and/or the co-administered medicinal product.
Eliminated by kidneys using a combination of glomerular filtration and active tubular secretion.
Renal clearance involves both glomerular filtration and active tubular secretion, moderated by(pH-dependent) tubular reabsorption.
Elimination of sitagliptin occurs primarily via renal excretion and involves active tubular secretion.
Studies have established the pathway of active tubular secretion of tenofovir to be influx into proximal tubule cell by the human organic anion transporters(hOAT) 1 and 3 and efflux into the urine by the multidrug resistant protein 4(MRP 4).
Adefovir is excreted renally, by a combination of glomerular filtration and active tubular secretion.
With medicinal products that reduce renal function or compete for active tubular secretion(e.g. cidofovir) may increase serum concentrations of emtricitabine, tenofovir and/or the co-administered medicinal products.
Emtricitabine is principally eliminated by the kidney via glomerular filtration and active tubular secretion.
Coadministration of Hepbest 25 mg with drugs that reduce renal function or compete for active tubular secretion may increase concentrations of tenofovir and other renally eliminated drugs and this may increase the risk of adverse reactions.
Emtricitabine is primarily excreted by the kidneys by both glomerular filtration and active tubular secretion.
Tenofovir is primarily excreted by the kidney by both filtration and an active tubular transport system(human organic anion transporter 1[hOAT1]) with approximately 70-80% of the dose excreted unchanged in urine following intravenous administration.
Tenofovir is eliminated from the body by the kidneys by both glomerular filtration and active tubular secretion.
Renal elimination of mirabegron is primarily through active tubular secretion along with glomerular filtration.
Renal function: Adefovir is excreted renally, by a combination of glomerular filtration and active tubular secretion.
Co-administration of emtricitabine with medicinal products that are eliminated by active tubular secretion may lead to an increase in serum concentrations of either emtricitabine or a co-administered medicinal product due to competition for this elimination pathway.
The clearance of metformin in healthy individuals is 400 ml/ min(4 times more than CC),which indicates active tubular secretion.
Since tenofovir is primarily eliminated by the kidneys,co-administration of tenofovir disoproxil with medicinal products that reduce renal function or compete for active tubular secretion via transport proteins hOAT 1, hOAT 3 or MRP 4(e.g. cidofovir) may increase serum concentrations of tenofovir and/or the co-administered medicinal products.
Emtricitabine and tenofovir are primarily excreted by the kidneys by a combination of glomerular filtration and active tubular secretion.
Meropenem is a substrate of OAT1 and OAT3 and as such,probenecid competes with meropenem for active tubular secretion and thus inhibits the renal excretion of meropenem.
Hydrochlorothiazide is not metabolised and is excreted almost entirely as unchanged drug by glomerular filtration and active tubular secretion.
Other medicinal products(including e.g. tenofovir)administered concurrently that compete with or inhibit active tubular secretion may increase aciclovir concentrations by this mechanism.
The renal clearance of levetiracetam and ucb L057 is 0.6 and 4.2 ml/min/kg respectively indicating that levetiracetam is excreted by glomerular filtration with subsequent tubular reabsorption andthat the primary metabolite is also excreted by active tubular secretion in addition to glomerular filtration.