Примери за използване на Amyloid plaques на Английски и техните преводи на Български
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Amyloid plaques, rampant cell death.
Look at the texture… the amyloid plaques.
Amyloid plaques were a mystery for many years.
You're not a spring chicken anymore,so you have accumulated some amyloid plaques with age.
That's what amyloid plaques are like when they form in body tissues.
Next Article» Alzheimer's disease andtype 2 diabetes- hope for inhibitors against amyloid plaques.
These amyloid plaques match those found in Alzheimer's, but their spatial distribution differs.
And when this happens, it binds to itself,forming sticky aggregates called amyloid plaques.
This suggests that the appearance of amyloid plaques isn't simply an unexpected consequence of developing CJD.
Specifically, there is a buildup of beta-amyloid,which is a protein that produces amyloid plaques.
This initial step into the disease,this presence of amyloid plaques accumulating, can already be found in your brains.
In this experiment,the increase in autophagy led to a decrease in the amyloid plaques and tau.
The accumulation of amyloid plaques in the brain is often used as a biomarker for Alzheimer's disease.
One of its key features is a buildup of so-called beta amyloid plaques inside brain cells.
Amyloid plaques, which are protein deposits, play a crucial role in the development of Alzheimer's disease and type 2 diabetes.
But research in recent years has revealed that people can have amyloid plaques without having dementia.
These amyloid plaques in the brain match those found in Alzheimer's dementia disease, but their spatial distribution varies.
Research in recent years has shown that people can have these amyloid plaques without having Alzheimer's.
They had decreased amounts of amyloid plaques and tau neurofibrillary tangles(both hallmarks of Alzheimer's disease) in their brains.
As a biologist explains, alpha secretase dominates in healthy neurons so that amyloid plaques do not accumulate in them.
Caffeine helps prevent the formation of amyloid plaques and neurofibrulary tangles in the brain- two hallmarks of Alzheimer's.
This makes sense, since blood brings oxygen andnutrients that feed brain cells and clear out amyloid plaques.
Effective therapeutics to counteract the formation of amyloid plaques in Alzheimer'sdisease and type 2 diabetes are not yet available.
Amyloid plaques are one of the first hallmarks of Alzheimer's disease and are thought to contribute to neurodegeneration by killing off brain cells.
In fact, studies have shown that curcumin actually suppresses the formation of the amyloid plaques linked with Alzheimer's disease.
Amyloid plaques are the result of the excess production and buildup of beta-amyloid, a fragment of the protein called“amyloid precursor protein.”.
The development of the neurofibrillary tangles and amyloid plaques in the brain that are characteristic of the disease have been associated with zinc deficiency.
It is thought that disturbed sleep patterns in patients with AD could be due to the growth of the amyloid plaques and resulting neuronal changes.
And studies have shown that this process can even lead to the formation of amyloid plaques, or Alzheimer's plaques, which are a hallmark of Alzheimer's disease.
The findings may lead to potential new therapeutic strategies, they say, butalso imply that there are limitations to therapies that are aimed at eliminating amyloid plaques from patient's brains.