Примери за използване на Nsaids can на Английски и техните преводи на Български
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NSAIDS can attenuate the antihypertensive effect of both.
Blood pressure- NSAIDs can increase blood pressure.
NSAIDs can cause a number of side effects, some of which may be very serious.
Aside from the precautions mentioned above, taking NSAIDs can have side effects.
Some NSAIDs can react unpredictably with other medicines.
Co-administration of loop diuretics and NSAIDs can result in a decreased natriuretic response.
Some NSAIDs can increase the risk of heart attacks on their own.
But, in addition, NSAIDs can reduce blood loss by about 30%.
Mifepristone: NSAIDs should not be used for8-12days after mifepristone administration as NSAIDs can reduce the effect of mifepristone.
In fact, NSAIDs can actually accelerate the degenerative process.
Therefore, the simultaneous administration of nicorandil with NSAIDs can increase the risk of hyperkalaemia due to a synergistic effect.
However, NSAIDs can reduce renal function in elderly patients.
Aspirin and other NSAIDs can cause hyperkalemia by inhibiting prostaglandin synthesis;
Mifepristone: NSAIDs should not be taken for 8-12 days after mifepristone administration, NSAIDs can reduce the effects of mifepristone.
As NSAIDs can cause troublesome side effects, alternatives are often recommended first.
Quinolone Antibiotics: Animal data indicate that high doses of quinolones in combination with NSAIDs can increase the risk of developing convulsions.
Additionally, some NSAIDs can adversely interact with aspirin, increasing the risk of bleeding even more.
Alcohol does not have an interaction with these particular painkillers,although drinking excessive amounts while using NSAIDs can irritate the gut and increase the risk of internal stomach bleeding.
Both colchicine(Colcrys) and NSAIDs can be used during an acute gout attack to decrease inflammation and pain.
NSAIDs can damage the liver, so you may need regular blood tests to check liver function.
Data available including a new epidemiological study(FVG GI study)confirms that all NSAIDs can induce damage to the gastroduodenal mucosa and increase the risk of upper gastrointestinal complications(UGIC).
NSAIDs can cause an increase in the blood levels of some liver enzymes but this is a rare event with the exception of aspirin.
Prolonged treatment with NSAIDs can reduce the diuretic and hypotensive effect of the drug in question, as well as lead to disruption of the kidneys.
NSAIDs can affect blood clotting but this response is not clinically significant except in children who already have a blood clotting abnormality.
NSAIDS can attenuate the antihypertensive effect of both angiotensin II antagonists and hydrochlorothiazide when administered simultaneously.
NSAIDs can also cause an increased risk of serious gastrointestinal adverse events especially in the elderly, including bleeding, ulceration, and perforation of the stomach or intestines, which can be fatal.
NSAIDs can cause clinically important damage of the gastrointestinal tract, increasing the incidence of bleeding in the upper gastrointestinal tract and of perforation, although serious complications and death are relatively infrequent.