Ví dụ về việc sử dụng Its receptor trong Tiếng anh và bản dịch của chúng sang Tiếng việt
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Its receptor is GPRC6A.
They stop the protein CGRP from getting into its receptor.
Circumstances, GH(growth hormone) binds to its receptor in the liver, and other tissues, and.
The toxin kills only cells that contain its receptor.
When fMLP binds to its receptor, it dissociates the G protein into its two components Gαi and Gβγ.
For example, both IGFBP-2 andIGFBP-5 bind IGF-1 at a higher affinity than it binds its receptor.
The absence of leptin(or its receptor) leads to uncontrolled hunger and resulting obesity.
In the treatment of Cushing's syndrome,Mifepristone blocks the binding of cortisol to its receptor.
Interleukin 6 and its receptor were discovered and cloned at Osaka University, Japan, by Tadamitsu Kishimoto in the 1980s.
This indicates that BQ-123can work as an antagonist to remove ET-1 from its receptor(ETA).[3].
The binding of TNF to its receptor and its displacement by LT confirmed the functional homology between the two factors.
In rheumatoid arthritis,infliximab seems to work by preventing TNF-α from binding to its receptor in the cell.
A team of Japanese scientists has identified a peptide and its receptor that direct lateral roots into growing with the right spacing.
Because of this competitive antagonism, tamoxifen acts like a key broken off in the lock that prevents any other key from being inserted,preventing estrogen from binding to its receptor.
The basic mechanism workssuch that PEG-interferon alpha-2b will bind to its receptor, interferon-alpha receptor 1 and 2(IFNAR1/2).
They analyzed the behavior of cultured cancer cells, when activated sandranol OR10H1, and identified a signaling pathway that is"turned on" in the cell,when the odorant binds to its receptor.
Scientists were researching CBD in the last few years andthe results are showing that its receptors are based in the central nervous system.
The activity of epidermal growth factor and its receptor, the EGFR, have been identified as key drivers in the process of cell growth and replication.
In unpublished test tube experiments, researchers found that MHCP mimics insulin,activates its receptor, and works synergistically with insulin in cells.
As such,SERMs are necessary to stop estrogen from binding to its receptor sites, thus instigating the chemical reactions necessary to lead to the fruition of these side effects as well as some of the positive elements associated with this hormone.
The pleiotropic effects of IL-2 are enabled due to the fact that IL-2 signal can be transduced via 3 different signaling pathways; JAK-STAT, PI3K/Akt/mTOR and MAPK/ERK pathway.[1]After IL-2 binding to its receptor, cytoplasmatic domains of CD122 and CD132 heterodimerize.
Evidence exists showing the presence of GnRH and its receptor in extrapituitary tissues as well as a role in progression of some cancers.[2].
CSGAGs interact with heparin binding proteins, specifically dermatan sulfate interactions with fibroblast growth factor FGF-2 and FGF-7 have been implicated in cellular proliferation and wound repair[15] while interactions with hepatic growth factor/scatter factor(HGF/SF)activate the HGF/SF signaling pathway(c-Met) through its receptor.
The main physiological action of GH is to promote growth anddevelopment, and its receptors is distributed in various tissues all over the body.
Mepolizumab binds to IL-5 and prevents it from binding to its receptor, more specifically the interleukin 5 receptor alpha subunit, on the surface of eosinophil white blood cells.
The most prominentis that it reversibly blocks the action of adenosine on its receptor and consequently prevents the onset of drowsiness induced by adenosine.
This suggests that it is able to block binding of dihydrotestosterone to its receptor targets in the prostate gland, but its partial agonist effects at androgen receptors prevent the side effects associated with the anti-androgenic therapies traditionally used for treatment of BPH.
While abciximab has a short plasma half-life,due to its strong affinity for its receptor on the platelets, it may occupy some receptors for weeks.
In particular, the PTPRF gene, which is known to suppress intracellular signals thatare usually triggered by insulin binding to its receptor on the cell surface, may serve as a biomarker linking insulin resistance with insufficient milk supply.