Exemplos de uso de Cell response em Inglês e suas traduções para o Português
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Otherwise, red blood cell response will be reduced.
Cell response to stimuli depends on a complex signaling process.
Bone that undergoes little stress does not promote osteogenesis, andlacking bone cell response.
Part of this cell response is brought on by inflammatory mediators such as chemotactic factors.
And as far as a cure, it doesn't look likethey're taking tissue samples, studying cell response.
Therefore, the antibody-mediated direct cell response contributes to the pathogenesis of immune TRALI.
Protein(s) produced due to the signal will be responsible for the kind of cell response.
Homocysteine also plays an essential role in the methylation process, a cell response that regulates the DNA and the genome as well.
To ensure cell response to cytotoxic and noncytotoxic agents, a positive and negative control was conducted for each microplate.
HLA molecules participate in antigen presentation and cell response playing an important role in the immune response. .
The pathophysiological mechanism is not fully understood yet, andis the result of a complex immune process mediated by Th1 and Th17 cell response.
The presence of immunocompetent cell response activated in the epidural interface of pulpous muscle hernation reinforces the concept of immunogenic capacity of this structure.
Melatonin is very important for the insulin balance,since a disorder in this system can deactivate the cell response to insulin and glucose.
However, no studies have definitively explained the molecular mechanism of cell response mediated by orthodontic expansion forces that leads to proliferation and differentiation of the progenitor cells into osteoblasts.
It is suggested that T. rubrum has the ability to suppress the expression of TLR receptors in keratinocytes andLC necessary for stimulation of Th1-type cell response.
In contrast, no significant changes in IL-4 or IL-10 were observed in either group,suggesting that the Th2 cell response and anti-inflammatory activity of these cytokines remained unchanged.
This means that differences in the structure of the extracellular matrix affect the biomechanical properties of the whole network in addition to the signals that transform cell response.
Several mechanisms of action have been described,such as the reduction in cell response and, less effectively, in humoral response and anti-inflammatory properties interference with the migration and action of neutrophils, lymphocytes and monocytes.
The authors observed more effective stimulation of fibroblast proliferation for cells treated with the 632.8nm laser,suggesting that the operating wavelength of the laser interfered with the cell response.
In 2 weeks the cell response is already moderate; in 4 weeks the fibroblasts appear; in 6 weeks the collagen deposition is intensified; in 8 weeks the chronic inflammatory cells are disposed along a solid collagen deposition.
The clinical presentations of leprosy are associated to distinct immunologic patterns,ranging from an immune response mediated by cells to M. leprae to the absence of cell response to the antigens of M. leprae.
The quickness of the hair cell response may also be due to the fact that it can increase the amount of neurotransmitter release in response to a change as little as 100 μV in membrane potential.
The main effector proteins involved in the fi brosis mechanisms in SSc are the cytokines, chemokines, and the family of ECM growth factors,which induce fibrogenic cell response by increasing the expression of genes encoding ECM constituents.
Since previous studies has suggested that oxidative stress,as part of the host cell response, might play an important role in the pathogenesis of a variety of rna viral infections, this study aimed to investigate whether this event can also be related to carv infection.
You might remember that when we speak about cell membrane receptors we can think about a kind of antenna receiving(chemical) signals from outside the cell andtransmitting them to the cell nucleus, where the cell response will be generated accordingly.
Bone graft take has 5 stages: 1 inflammatory,promoting a host cell response; 2 tissue revascularization; 3 osteoconduction, in which the graft has the function of being a framework for the growth of vessels and bone formation; 4 osteoinduction, the host's mesenchymal cells are induced to turn into osteoblasts by proteins found in the graft and 5 bone remodeling with the characteristics of continuous bone formation and resorption.
This field of science is based on the interplay of three essential components: responsive cells embryonic or adult stem cells, morphogens molecules of protein nature capable of inducing cell response and scaffolds structures designed to mimic the extracellular matrix and serve as a physical support for the cells development.
Normal human chondrocytes were procured from Promocell, Germany and were cultured in chondrocyte growth medium Promocell, Germany under standard laboratory conditions humidified atmosphere of 95% air and 5% CO2, 37 °C. Monolayer culture of chondrocytes has long been used as a method for assessing in vitro cell response to treatment.
Imatinib, an inhibitor of c-Abl, reduces fi brotic responses in animal models andhave shown to be promising in the treatment of patients with SSc. Also relevant as mediators of profibrotic cell response to TGF? are genes from the early growth response EGR family, particularly EGR-1, whose expression is induced by chemical or mechanical tissue injury and whose levels are elevated in skin lesions of SSc, as demonstrated in a murine model of bleomycininduced scleroderma.
These clinical forms, known as polar forms, are classified into: tuberculoid leprosy in immuneresistance Th1 pole, when it has few lesions orisolated neural affections, positive cell response to intradermoreaction of Mitsuda IRM, and when there are bacilli in cutaneous lesions or nerves; and as Virchowian Hansen disease WH when it has many lesions, exacerbated humoral response, negative IRM and many bacilli in lesions and nerves, forming the non-responsive immune pole to the bacillus.