Exemplos de uso de Perfusional em Inglês e suas traduções para o Português
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Or perfusional(first-pass perfusion) alterations.
One patient presented a hepatic punctiform calcification,with no associated perfusional alteration and identified only in the second analysis.
Like in cirrhosis, perfusional alterations in patients with metastasis show contrast uptake in the hepatobiliary phase, differently from metastases.
The growing familiarity with the off-pump coronary artery bypass grafting technique has showed that there in no need for perfusional support 12, 14.
Perfusion study(A) during hyperemia induced by dipyridamole identified perfusional deffect(asterisk) in the region of the anterior descending coronary artery black area.
Amongst the cases presented to the gastroenterologist, the changes in the diagnosis were associated to steatosis, cholelithiasis andhepatic calcifications without perfusional alterations.
It is important to remember that the presence of liver calcification without textural or local perfusional changes should be considered as being of probable residual origin.
It is through the reactive hyperemia that perfusional balance returns in the sinusoids of the transplanted liver, resulting in lower elevation of aminotransferases and bilirubin.
In complete agreement with the visual analysis results,the automatic detection technique indicates a reversible perfusional defect in the apical region and postero-lateral wall.
In order to evaluate perfusional viability, the statistically significant difference between the scores of the myocardial segments at the stress and rest and rest and redistribution images was analyzed.
For the 13 patients evaluated in the present study, myocardial viability at thallium-201 was considered in the presence of perfusional reversibility between the perfusion phases, regardless the uptake degree.
Perfusional alterations present a signal similar to the one of the remainder hepatic tissue during the portal and hepatobiliary phases, while most HCCs, except the well-differentiated ones, present hyposignal in the hepatobiliary phase.
Accordingly with the visual analysis performed by two experienced observers,the results indicate a reversible, extensive perfusional defect on the anterior, septal, apical and postero-lateral walls of the myocardium.
Amongst the 100 cases evaluated, the 18 patients who had their secondary diagnoses modified presented steatosis, nephrolithiasis, adrenal nodules, renal cysts andhepatic calcifications without perfusional alterations.
Diffuse increased perfusional states can be seen in normal individuals, global cerebral hyperperfusion complicating embolic stroke, carotid endarterectomy with the post-CEA hyperperfusion syndrome, traumatic head injury, and in the setting of hypercapnia.
The absence of clinical or neurological symptoms of ischemia in the majority of patients with unilateral or bilateral agenesis or hypoplasia of the ICA leads to the assumption that,most of times, perfusional deficit is not present.
In this context, the evaluation of the fetal brain vascularization andblood flow is extremely relevant, considering that perfusional deficits in this territory may be determinant in a poor prognosis for the development of the central nervous system and even for the fetal welfare.
Eighteen 18% of the 100 patients had their secondary diagnosis modified, although without statistical significance p 0.143, as follows: steatosis 7%, adrenal nodule 1%,nephrolithiasis 7%, renal cysts 2% and hepatic calcifications without perfusional alterations 1% Figures 2, 3, 4 and 5.
We may also use stressor agents such as dobutamine, adenosine, regadenoson, anddipyridamole to assess contractile cine MRI or perfusional first-pass perfusion alterations, respectively, or even perform combined protocols for multimodal assessment of the myocardium.
Thus, the hepatobiliary contrast agents provide,initially, perfusional data similar to those from nonspecific extracellular gadolinium with renal excretion, and later, hepatocyte-selective data with biliary excretion, thus allowing for the differentiation between lesions containing hepatocytes and lesions without functional hepatocytes.
Hemangiomas also may be exophytic or pedunculated, andnot infrequently present small, adjacent arterial portal venous shunt determining transitory perfusional disorder in the adjacent hepatic parenchyma observed at CT and MRI.
High resolution CT demonstrated details of the parenchymal alterations particularly characterized by predominantly cystic bronchiectasis clustered in the central regions,diffuse perfusional alterations in the pulmonary parenchyma mosaic pattern of attenuation, besides increased caliber of the trachea and of the main bronchi which also presented jagged contours corresponding to tracheobronchial diverticulosis Figure 2.
Correlating nephrograms with right renal arteries stenoses[14.2% n 8], and left renal arteries stenoses[25.45% n 14], we have observed that the higher the stenosis level is, independently from topography,the higher the probability of finding a perfusional alteration that could identify hemodynamically significant alterations.
Several independent studies conducted by various investigators using perfusion myocardial scintigraphy demonstrated the occurrence of remarkable perfusional disorders in the hearts of patients with chronic Chagas. In those studies, the coronary arteries were angiographically normal, and the results vehemently attest the possibility of microvascular alterations, according to experimental studies.
In order to evaluate the feasibility and potential of this method as an aid in the detection of myocardial perfusion defects, rest/stress images of one adult man andone adult woman with reversible, moderate perfusional defects were selected for statistical comparison with the template images.
However, 123-iodine singlephoton emission computed tomography SPECT can be utilized, andhas already been utilized to demonstrate the absence of areas with perfusional deficit, considering that some few patients have experienced episodes of transitory ischemic accidents of unidenfied etiology.
IHD was diagnosed in the presence of angina or possible myocardial infarction according to the Word Health Organization WHO cardiovascular questionnaire and/or compatible electrocardiographic changes Minnesota Code: Q and QS[1.1-2, 1.3] patterns; S-T[J] junction and segment depression[4.1-4]; items of the T[5.1-3] wave, and complete left bundle block[7.1]and/or perfusional abnormalities fixed or variable in myocardial scintigraphy at rest and after dipyridamol.