Exemplos de uso de Tracheobronchial tree em Inglês e suas traduções para o Português
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Lymph node enlargement with compression of the tracheobronchial tree.
In the normal tissue of the tracheobronchial tree the BMP-2 protein is not present.
Our case series showed that 37 FBs 92.5% were lodged in the tracheobronchial tree.
The development of the tracheobronchial tree begins on the 24th day of embryogenesis.
In addition it serves as a route by which microorganisms can enter the tracheobronchial tree.
The nasal cavities, larynx, and tracheobronchial tree were assessed.
In the tracheobronchial tree, the most common manifestation in our patients was inflammation.
A method developed for the study of the tracheobronchial tree is fiberoptic bronchoscopy.
It is characterized by recurring expectoration of peculiar molds from the tracheobronchial tree.
This vibration is transmitted to the patient rib cage and tracheobronchial tree, displacing the secretions and facilitating expectoration.
As a rule,coughing attacks in a child arise from irritation of the receptors of the tracheobronchial tree.
The presence of an asymptomatic congenital anomaly of the tracheobronchial tree in thoracic surgery patients can be challenging not only to thoracic surgeons but also to anesthesiologists.
It usually involves communication between the pericardium and the tracheobronchial tree or digestive tract.
Blood flooding the tracheobronchial tree can originate from the vascular network spread throughout the lung tissue, that is, from the bronchial arterial circulation and from the pulmonary arterial circulation.
Inside the lungs, the bronchi divide into smaller bronchi,forming the branches of the tracheobronchial tree.
Since ELS, which is less frequently encountered than is ILS,presents no contact with the tracheobronchial tree, it is rarely accompanied by clinical symptoms, and is more commonly associated with other congenital malformations.
This can cause significant obstruction by the formation of branching gelatinous casts in the tracheobronchial tree.
The inhaled spores deposit on the mucosaof the paranasal sinuses, larynx, and the tracheobronchial tree, and the dark airway cavities favor their growth as hyphae.
Therefore, FEV1 and FVC both also reflect changes in flow,albeit in different segments of the tracheobronchial tree.
However, entering of the foreign body can be caused by erosion in the esophagus or tracheobronchial tree after the aspiration or ingestion of foreign bodies such as teeth, dentures, needles, pins and others. 1,3,6.
Therefore, they should be included in the differential diagnosis of profiles of tracheobronchial tree obstruction.
Direct laryngoscopy did not show the presence of foreign body in the larynx or the tracheobronchial tree, and then submitted to Rigid Esophagoscopy and viewed the foreign body in the middle third of the esophagus, carrying their withdrawal uneventful.
Apparently, the typical uncomplicated benign infections affect only the mucosa of the upper tracheobronchial tree.
High concentrations of NO in the tracheobronchial tree induce hyperemia, plasma exudation, secretion of mucus and lymphocyte proliferation TH2, responsible for eosinophilic proliferation in a sequence of events that characterize inflammatory phenomena.
Perform a white light bronchoscopy(WLB)inspection in the standard fashion examining the tracheobronchial tree to the segmental level.
Although endotracheal polyps are rarely found,they should be included in the differential diagnosis of partial or complete obstruction of the tracheobronchial tree.
Our study identified lower airway inflammation in four patients 26.7%, ulceration and"cobblestone" mucosa in the tracheobronchial tree in four patients 26.7%, and bronchial stenosis in three patients 20.
In general, when such an abnormality occurs precociously, mediastinal cysts are formed,usually presenting no communication with the tracheobronchial tree.
Airway contact with cigarette smoke induces changes in the respiratory system, such as mucus hypersecretion,deficit in mucociliary transport, tracheobronchial tree defects, small airway restriction accompanied by increased closing capacity, and a trend toward changes in the ventilation-perfusion ratio.
Pulmonary sequestration is characterized by a mass of nonfunctioning lung tissue that does not communicate with the tracheobronchial tree.