Examples of using Reverse mutation in English and their translations into Croatian
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Colloquial
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Computer
Then reverse mutation is possible?
Crizotinib was not mutagenic in vitro in the bacterial reverse mutation(Ames) assay.
Reverse mutation is induced by infusion of an antitoxin, concentrated doses of it.
Cabazitaxel did not induce mutations in the bacterial reverse mutation(Ames) test.
In an in vitro bacterial reverse mutation assay(Ames test), increases in the number of revertant colonies were observed in 2 of the 5 strains exposed to florbetapir.
Eribulin was not mutagenic in vitro in the bacterial reverse mutation assay Ames test.
Trifluridine was shown to be genotoxic in a reverse mutation test in bacteria, a chromosomal aberration test in mammal-cultured cells, and a micronucleus test in mice.
Rilpivirine has tested negative in the absence andpresence of a metabolic activation system in the in vitro Ames reverse mutation assay and the in vitro clastogenicity mouse lymphoma assay.
A bacterial reverse mutation assay study(“Ames test”) was performed with the cysteamine bitartrate used for PROCYSBI and cysteamine bitartrate did not show any mutagenic effects in this test.
Peginterferon beta-1a was not mutagenic when tested in an in vitro bacterial reverse mutation(Ames) test and was not clastogenic in an in vitro assay in human lymphocytes.
Temsirolimus was not genotoxic in a battery of in vitro(bacterial reverse mutation in Salmonella typhimurium and Escherichia coli, forward mutation in mouse lymphoma cells, and chromosome aberrations in Chinese hamster ovary cells) and in vivo(mouse micronucleus) assays.
Maraviroc was not mutagenic or genotoxic in a battery of in vitro and in vivo assays including bacterial reverse mutation, chromosome aberrations in human lymphocytes and rat bone marrow micronucleus.
Trametinib was not genotoxic in studies evaluating reverse mutations in bacteria, chromosomal aberrations in mammalian cells and micronuclei in the bone marrow of rats.
Lopinavir/ritonavir was not found to be mutagenic or clastogenic in a battery of in vitro andin vivo assays including the Ames bacterial reverse mutation assay, the mouse lymphoma assay, the mouse micronucleus test and chromosomal aberration assays in human lymphocytes.
Bazedoxifene was not genotoxic or mutagenic in a battery of tests,including in vitro bacterial reverse mutation assay, in vitro mammalian cell forward mutation assay at the thymidine kinase(TK±) locus in L5178Y mouse lymphoma cells, in vitro chromosome aberration assay in Chinese hamster ovary(CHO) cells, and in vivo mouse micronucleus assay.
Darunavir was not mutagenic or genotoxic in a battery of in vitro andin vivo assays including bacterial reverse mutation(Ames), chromosomal aberration in human lymphocytes and in vivo micronucleus test in mice.
Carfilzomib was not mutagenic in the in vitro bacterial reverse mutation(Ames) test and was not clastogenic in the in vivo mouse bone marrow micronucleus assay.
Naltrexone was negative in the following in vitro genotoxicity studies:bacterial reverse mutation assay(Ames test), the heritable translocation assay, CHO cell sister chromatid exchange assay, and the mouse lymphoma gene mutation assay.
Genotoxicity studies demonstrated that clofarabine was not mutagenic in the bacterial reverse mutation assay, but did induce clastogenic effects in the non-activated chromosomal aberration assay in Chinese Hamster Ovary(CHO) cells and in the in vivo rat micronucleus assay.
Tedizolid phosphate was negative for genotoxicity in all in vitro assays(bacterial reverse mutation[Ames], Chinese hamster lung[CHL] cell chromosomal aberration) and in all in vivo tests mouse bone marrow micronucleus, rat liver unscheduled DNA synthesis.
Ritonavir was not found to be mutagenic or clastogenic in a battery of in vitro andin vivo assays including the Ames bacterial reverse mutation assay using S. typhimurium and E. coli, the mouse lymphoma assay, the mouse micronucleus test and chromosomal aberration assays in human lymphocytes.
In genotoxicity studies with other recombinant growth hormone preparations, there was no evidence of gene mutation in bacterial reverse mutation assays, chromosomal damage in human lymphocyte and mouse bone marrow cells, gene conversion in yeast or unscheduled DNA synthesis in human carcinoma cells.
But in the case ofthis one, this one that we borrowed from Tibet,a genetic mutation reversed the equation.
We cannot reverse our mutations without the serum, nor can the Aquans benefit from the knowledge of their ancestors.
I have failed in isolating and reversing the mutations.
Our only purpose here is to find the means of reversing our mutations.