Examples of using Mitochondrial dysfunction in English and their translations into Danish
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Another thing that contributes to mitochondrial dysfunction is latent viral infection such as the ones of the herpes family.
Ketone bodies production through intermittent fasting andthe ketogenic diet is the most promising treatment for mitochondrial dysfunction.
Intracellular viruses andbacteria can lead to severe mitochondrial dysfunction and ketosis remains by far our best chance against them.
Mitochondrial dysfunction contributes to congestive heart failure, type 2 diabetes, autoimmune disorders, aging, cancer, and other diseases.
Have clinical and laboratory follow-up andshould be fully investigated for possible mitochondrial dysfunction in case of relevant signs or symptoms.
There have been reports of mitochondrial dysfunction in HIV negative infants exposed in utero and/ or postnatally to nucleoside analogues.
Understanding these relationships will help to understand the underlying etiology for a range of diseases linked with mitochondrial dysfunction, such as diabetes and obesity.
Mitochondrial dysfunction has wide-ranging implications, as the health of the mitochondria intimately affects every single cell, tissue and organ within your body.
They are positively influenced by adiet that is non-glycating, i.e. a low carb diet as opposed to a high carb diet which induces mitochondrial dysfunction and formation of reactive oxygen species.
There have been reports of mitochondrial dysfunction in HIV-negative infants exposed in utero and/ or post-natally to nucleoside analogues.
Viread may also cause lactic acidosis(a build-up of lactic acid in the body) and, in the babies of mothers taking Viread during pregnancy, mitochondrial dysfunction damage to the energy-producing components within cells that can cause problems in the blood.
Mitochondrial dysfunction: nucleoside and nucleotide analogues have been demonstrated in vitro and in vivo to cause a variable degree of mitochondrial damage.
Members of the herpes virus family(i.e. cytomegalovirus and Epstein-Barr virus which most people have as latent infections!), can go after our mitochondrial DNA,causing neurodegenerative diseases by mitochondrial dysfunction.
There have been reports of mitochondrial dysfunction in HIV-negative infants exposed in utero and/ or post-natally to nucleoside analogues see also section 4.8.
Any child exposed in utero to nucleoside and nucleotide analogues, even HIV-negative children, should have clinical and laboratory follow-up andshould be fully investigated for possible mitochondrial dysfunction in case of relevant signs or.
There are a variety of applications for this new methodology, with mitochondrial dysfunction is implicated in many human diseases, such as Diabetes mellitus7, obesity8, multiple sclerosis9, Parkinson's diseases10, Alzheimer's disease11 and some type of cancers12.
Any child exposed in utero to nucleoside and nucleotide analogues, even HIV-negative children, should have clinical and laboratory follow-up andshould be fully investigated for possible mitochondrial dysfunction in case of relevant signs or symptoms.
Mitochondrial dysfunction: review of the postmarketing safety database shows that adverse events indicative of mitochondrial dysfunction have been reported in the neonate and infant population exposed to one or more nucleoside analogues see also section 4.4.
Any child exposed in utero to nucleoside and nucleotide analogues, even HIV negative children, should have clinical and laboratory follow-up andshould be fully investigated for possible mitochondrial dysfunction in case of relevant signs or symptoms.
Understanding these relationships will help to understand the underlying etiology for a range of diseases linked with mitochondrial dysfunction, such as diabetes and obesity. Recent advances in instrumentation, has enabled the monitoring of distinct parameters of mitochondrial function in cell lines or tissue explants.
Any child exposed in utero to nucleoside and nucleotide analogues, even HIV negative children, should have clinical and laboratory follow-up andshould be fully investigated for possible mitochondrial dysfunction in case of relevant signs or symptoms.
There are a variety of applications for this new methodology, with mitochondrial dysfunction is implicated in many human diseases, such as Diabetes mellitus7, obesity8, multiple sclerosis9, Parkinson's diseases10, Alzheimer's disease11 and some type of cancers12. Importantly, our work is performed in vivo, where all environmental influences- such as cytokines, development-related growth etc- are active, thereby providing a physiologically relevant view in vivo respiration and metabolic profile.
As with all NRTIs, Trizivir may also cause lactic acidosis(a build-up of lactic acid in the body)and, in the babies of mothers taking Trizivir during pregnancy, mitochondrial dysfunction damage to the energy-producing components within cells that can cause problems in the blood.
As with all other NRTIs, Zerit may also cause lactic acidosis(a build-up of lactic acid in the body) and, in the babies of mothers taking Zerit during pregnancy, mitochondrial dysfunction damage to the energy-producing components within cells that can cause problems in the blood.
As with all other NRTIs, Epivir may also cause lactic acidosis(a build-up of lactic acid in the body) and,in the babies of mothers taking Epivir during pregnancy, mitochondrial dysfunction damage to the energy-producing components within cells that can cause problems in the blood.
In other diseases, defects in nuclear genes lead to dysfunction of mitochondrial proteins.