Examples of using Mitochondrial dysfunction in English and their translations into Portuguese
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Mitochondrial dysfunction.
Reported a common mitochondrial dysfunction in this syndrome.
Mitochondrial dysfunction following exposure in utero.
There is also the accumulation of intramyocellular fat and mitochondrial dysfunction.
Mitochondrial dysfunction has been identified as one of the probable factors of dm.
Also, quinolinic acid plays a role in mitochondrial dysfunction in neurons.
Mitochondrial dysfunction has been associated with several cardiac, metabolic and neurodegenerative disorders.
Conclusion: The physical performance can be hindered by oxidative stress and mitochondrial dysfunction.
 Mitochondrial dysfunction is thought to be caused by environmental toxins and could be related to autism.
The brain has a high metabolic rate andneuronal functions are particularly sensitive to mitochondrial dysfunction.
The pathological processes involved in pd are mitochondrial dysfunction, oxidative stress, inflammation and excitotoxicity.
Simultaneous citaa3 and StO2 measurements allow for the detection of cellular dysoxia secondary to mitochondrial dysfunction.
Mitochondrial dysfunction appears as an early event in Alzheimer's disease(AD) pathology and in other neurodegenerative diseases.
Benign symmetrical lipomatosis is usually a sporadic illness,probably derived from mitochondrial dysfunction.
There have been reports of mitochondrial dysfunction in infants exposed in utero and/or post-natally to nucleoside analogues.
The pathophysiological process is characterized by oxidative stress, mitochondrial dysfunction, cytotoxicity and apoptosis.
There have been reports of mitochondrial dysfunction in HIV negative infants exposed in utero and/ or postnatally to nucleoside analogues.
The mechanism of neurodegeneration in hd seems to be related to excitotoxicity, oxidative stress and mitochondrial dysfunction.
However, while some parasites die due to mitochondrial dysfunction, others can resist nutritional stress and survive.
Skeletal muscle contributes directly to the development and progression of metabolic disorders,especially as a result of mitochondrial dysfunction.
There have been reports of mitochondrial dysfunction in HIV negative infants exposed in utero and/ or postnatally to nucleoside analogues.
In general, the effect of 3-np associated with the same capacity to cause mitochondrial dysfunction and generating reactive species.
Mitochondrial dysfunction may be a key underlying mechanism for occurrence of metabolic disease and diabetes; thus elucidating how this process occurs is of great value.
These characteristics are important because of mitochondrial dysfunction and increased levels of inflammatory cytokines, that are present after menopause.
This includes rapid depletion of atp, ca2+ release from intracellular stores, loss of ionic homeostasis, excitotoxicity,activation and mitochondrial dysfunction.
In animal models, it is able to prevent the cerebral and hepatic mitochondrial dysfunction induced by diabetes, associated with a reduction in oxidative stress, LDL-c and lipid peroxidation.
Introduction: ataxia telangiectasia(a-t) is a neurodegenerative disease,which evolves immunodeficiency in varying degrees, mitochondrial dysfunction and oxidative stress.
Damaged Mt DNA,its resultant mitochondrial dysfunction along with dysfunctions by and from immune and hypersensitivity reactions(CHz) are all associated with sudden deaths.
Increased intracellular calcium and ROS open mitochondrial channels,causing swelling and mitochondrial dysfunction, with subsequent evolution to cellular necrosis.
Besides, mitochondrial dysfunction results in ATP depletion and in turn is expected to have a major impact on intracellular Ca homeostasis.