Examples of using Thyroxin in English and their translations into German
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Free thyroxin- more accurate analysis.
A man is prescribed a synthetic analogue of thyroxin.
During life, thyroxin is relatively stable.
Patients are prescribed replacement therapy with synthetic thyroxin.
Analysis of thyroxin- the main test of A-cells.
A cells secrete thyroid hormones(thyroxin, triiodothyronine);
Many athletes also recycle the drug with growth hormone and thyroxin.
Secretion of thyroxin is subject to natural rhythms circadian and annual.
A-cells produce iodine-containing hormones thyroxin and triiodothyronine.
But mostly the hormone is formed in other tissues through the deiodination of thyroxin.
The patient is recommended to donate blood for thyroxin, triiodothyronine, antibodies.
Indications for analysis and preparation rules are the same as for thyroxin.
However, one should take into account that the surplus of thyroxin is as dangerous as its deficiency.
The most common other drugs are Masteron, Equipose, Oxandrolone,GH and thyroxin.
For example, the hormone thyroxin is responsible for metabolism, body tone and other important factors.
There is no evidence suggesting Zotrim cannot be taken alongside Thyroxin.
Hormones as Thyroxin(T4), Triiodthyronine(T3) and Calcitonin are produced out of the precursor protein thyreoglobulin Tg.
The main reason for the development of this type of bradycardiais the destruction of thyroid cells with a decrease in thyroxin release hypothyroidism.
Selenium catalyzes the conversion of Thyroxin(T4), the inactive precursor of the thyroid hormone, to the active form triiodothyronine T3.
If hypothyroidism is found in a patient during an additional examination,then he is immediately prescribed both potassium iodide and synthetic thyroxin.
Thyroxin is responsible for the various metabolic activities and the under production of this hormone results in several discomforts of the body.
In the thyroid gland two iodine-containing hormone- thyroxin(T4) and triiodothyronine(T3) and one peptide hormone- calcitonin are synthesized.
The thyroxin seperated from blood competes with a thyroxin bound to a glucose-6-phosphat dehydrogenase to form a specific connector.
The causes of thyrotoxic crisis are not only in the release of thyroxin and triiodothyronine, but also in the concomitant insufficiency of the adrenal glands.
Other adverse drug reactions may be considered somatropin classeffects, such as possible hyperglycaemia caused by decreased insulin sensitivity, decreased free thyroxin level and benign intra-cranial hypertension.
Concentration of free thyroxin(FT4) post baseline< LLN was reported in 8.0% of patients treated with Stivarga and 6.6% of patients receiving placebo.
It is a synthetic sodium salt of thyroxin and, for convenience, dosing is available in tablets from 50 to 150 mg the intervals between dosages are 25 mg each.
When there is an insufficient amount of thyroxin in the body(this is called hypothyroidism), the metabolic processes are interfered with and the result is a reduction in the optimal functioning of the body organs and system.
Barbiturates, salicylates and penicillin displaces thyroxin from the albumin bindings and the bindings on TBG are competitive inhibited from salicylates, heparin, diazepam, sulfonylurea, phenylbutazone and diphenylhydantoin.
When there is an insufficient amount of thyroxin in the body(this is called hypothyroidism), the metabolic processes are interfered with and the result is a reduction in the optimal functioning of the body organs and system.