Examples of using Atrioventricular node in English and their translations into Indonesian
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A large branch supplies the atrioventricular node.
Atrioventricular node ablation: Prevents the atria from sending electrical impulses to the ventricles.
Adenosine slows conduction of electrical impulses through the atrioventricular node.
The atrioventricular node functions as a gate, limiting the electrical activity that reaches the ventricles.
AVNRT occurs when a reentrant circuit forms within or just next to the atrioventricular node.
The electrical network serving the ventricles leaves the atrioventricular node through the Right and Left Bundle Branches.
Patients with WPWS have an accessory pathway that connects the atria and ventricles,in addition to the atrioventricular node.
The impulse reaches the atrioventricular node from where it reaches the ventricles through the atrioventricular bundle.
The pulse thengets“held up” in another set of cells called the Atrioventricular node, or AV node for short.
The atrioventricular node- the electrical connection between the atria and the ventricles- is bombarded with impulses trying to get through to the ventricles.
Death actually occurs from tissue damage,necrosis in the heart itself particularly in the sinus and atrioventricular nodes.
The atrioventricular node- the electrical connection between the atria and the ventricles- is bombarded with impulses trying to get through to the ventricles.
The rhythm of the heartbeat is normally controlled by the atrioventricular node, which produces an initial electrical impulse trigger each heartbeat.
Death actually occurs from tissue damage,necrosis in the heart itself… particularly in the sinus and atrioventricular nodes.
The impulse then travels to the center of your heart, to the atrioventricular node, which lies on the pathway between your atria and your ventricles.
The atrioventricular node in turn sends an impulse through the nerve network to the ventricles, initiating the same wave-like contraction of the ventricles.
In first-degree AV block,the impulse conducting from atria to ventricles through the atrioventricular node(AV node) is delayed and travels slower than normal.
From here, the impulse exits the atrioventricular node and travels through your ventricles, causing them to contract and pump blood throughout your body.
Its function is controlled by two impulse-generating tissues, situated in the heart itself,known as sinoatrial node and atrioventricular node.
It generates nerve impulses that spread to the atrioventricular node, where the impulses are amplified and spread to other regions of the heart by nerves called Purkinje fibers.
This pathway may allow electrical current to pass between the atria andthe ventricles without passing through the atrioventricular node, leading to short circuits and rapid heartbeats.
Tawara's discovery of the atrioventricular node prompted Arthur Keith and Martin Flack to look for similar structures in the heart, leading to their discovery of the sinoatrial node several months later.
In individuals with WPW syndrome, the electrical activity that starts in the sinus node travels through the accessory pathway as well as across the atrioventricular node to activate the ventricles through both paths.
The electrical impulse travels from the sinus node to the atrioventricular node(also called AV node), where impulses are slowed down for a very short period, then continue down the conduction pathway via the bundle of His into the ventricles.
The antiarrhythmic effects of beta blockers arise from sympathetic nervous system blockade-resulting in depression of sinus node function and atrioventricular node conduction, and prolonged atrial refractory periods.
The electrical impulsesthen arrive at a cluster of cells called the atrioventricular node(AV node)- usually the only pathway for signals to travel from the atria to the ventricles.
The criteria to diagnose a right bundle branch block on the electrocardiogram: The heart rhythm must originate above the ventricles(i.e., sinoatrial node, atria or atrioventricular node) to activate the conduction system at the correct point.
In cardiac uses, it works as a nonselective muscarinic acetylcholinergic antagonist, increasing firing of the sinoatrial node(SA)and conduction through the atrioventricular node(AV) of the heart, opposes the actions of the vagus nerve, blocks acetylcholine receptor sites, and decreases bronchial secretions.
The main pumping chamber, the ventricle, is protected(to a certain extent) against excessively high ratesarising from the supraventricular areas by a'gating mechanism' at the atrioventricular node, which allows only a proportion of the fast impulses to pass through to the ventricles.