Examples of using Mtdna in English and their translations into Malay
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I am wondering if you mistakenly referred to mtDNA in this statement.
The A and X mtDNA Haplogroups are predominant among the American Indians.
PrimPol is required for replication reinitiation after mtDNA damage.
Of all Ashkenazi-Jewish mtDNA is K, consisting of 4 major subtypes.
A recent study ofAfrican dogs found a high level of mtDNA diversity.
The mtDNA of the Ashkenazi Jews is classed as Near Eastern, but we need to look at the structure.
This was the original progression of the mtDNA of the M supergroup after the Flood.
A small amount of DNA can also befound in the mitochondria called as mitochondrial DNA or mtDNA.
Mitochondrial DNA(mtDNA) is more likely to get damaged in the course of everyday living.
A small portion of DNA can also be found in the mitochondria andis called mitochondrial DNA or mtDNA.
The mitochondria(mtDNA) act as cellular power plants and supplies energy to protein formation to work.
In mitochondria, a small amount of DNA can also be found andthere it is called mitochondrial DNA or mtDNA.
All mtDNA Haplogroups are subdivisions of L, then M and N and subsequently R, which itself is a mutation of Hg N.
However, its ancestry reports aren't nearly as substantial as 23andMe's,and doesn't include mtDNA or Y-DNA analysis.
This mtDNA comes from a common maternal ancestor who lived approximately 200,000 years ago(estimates vary).
Thus, the settlement of the Americas was late, with the YDNA and mtDNA mutations being of the second to last forms.
As we saw with the mtDNA, all other groups came from Hg L3, which split into two main groups M and N.
Now these are the findings based on the mathematical models that do notrecognise faster YDNA mutation due to mtDNA exposure.
There is a very small mtDNA M in Australia, with a similarly small percentage of mtDNA N in Papua New Guinea.
Haplogroup B moved south to Papua New Guinea and is in greatest concentrations among the Polynesians,being over 90% of their mtDNA.
That assumption is based on the premise that mtDNA does not force mutation of the Human Genome and that assumption is now being shown to be false.
This has proven to be beneficial in preventing damage to mitochondria membranes and mtDNA, which could directly reduce energy production.
We will also find that when we look at the mtDNA record for the female lines we are then able to place the female divisions in line with the YDNA male divisions.
Their base structure does not argue for a long occupation but rather an isolated DNA system thatsuffered no forced mutation by intermarriage with other mtDNA Haplogroups.
As seen from the Pasteur Institute research, mtDNA causes mutation where damage has occurred through disease, and also radiation, as we are now discovering.
The mtDNA is determined by reporting the polymorphic site such as for example 311C, meaning a mutation has occurred at base pair 16,311 and the base that changed here was actually changed to cytosine.
There is an argument that the YDNA K and the mtDNA P may have been there with the Papuans when the Tasmanian Aborigines came and in fact preceded the Australian Aboriginal YDNA C and mtDNA N.
The mtDNA is measured, but the effects of the female X chromosome on the male YDNA structure is considered important and it will be argued that it causes mutations in the YDNA structure at a much faster rate than the current model is believed to allow.
There is thus nothing in the mtDNA variations and Haplogroups to preclude the Bible story and the Genesis account being accommodated by, or accommodating, the scientific advances we find here.
Generality Mitochondrial DNA, or mtDNA, is the deoxyribonucleic acid that resides inside the mitochondria, ie the organelles of eukaryotic cells responsible for the very important cellular process of oxidative phosphorylation.