Examples of using Vasoconstriction in English and their translations into Vietnamese
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Vasoconstriction by adrenalone may lead to local necrosis.[1].
One of the primary concerns ofFDA is the ability of the drug to cause vasoconstriction.
Causes vasoconstriction of the mucous membranes, abdominal organs and skeletal muscles. Increases blood pressure.
When blood vessels narrow, this is called vasoconstriction, and it can affect body temperature.
It causes vasoconstriction which is common during periods when the temperatures in the atmosphere are low.
This is due to the presence in the tile of caffeine and phenylethylamine,which cause vasoconstriction.
Vasoconstriction of the muscular wall of the bronchus in the lungs is what triggers this cough reaction[citation needed].
It has beenclinically proven that bark extract contributes to vasoconstriction and increases blood clotting.
Vasoconstriction- the blood vessels in the nose would constrict so that less blood flow would lead to less nasal discharge.
Well, DMAA boosts your energy through vasoconstriction, a process which involves the restriction of the flow of your blood.
But several of his arteries did appear to narrow significantly,a condition called reversible cerebral vasoconstriction syndrome(RCVS).
At higher doses,it can cause raised arterial blood pressure, vasoconstriction(including coronary vasospasm) and bradycardia or tachycardia.
Users also ought to understand that unlike many other pre-workout supplements that contain nitric oxide,DMAA causes vasoconstriction of veins.
This is due to the unusual side effect of coronary vasoconstriction due to serotonin 5HT1B antagonism, which can precipitate a heart attack in those already at risk.
However, several of his arteries appeared to be significantly narrowed,a brain condition known as reversible cerebral vasoconstriction syndrome(RCVS).
Isometheptene is a sympathomimetic drug that works by inducing vasoconstriction, causing constriction of cerebral blood vessels and reducing migraine symptoms.
Nicotine causes vasoconstriction, a narrowing of the blood vessels that can limit the oxygenation of blood flow in the small vessels of the face or other parts of the body.
As the blood vessels tighten to minimize the bleeding,in a process known as vasoconstriction, both threats are averted by forming a blood clot.
The potent venodilating action of Pimobendan makes it a suitable treatment option for patients with congestive heart failure(CHF)as it also improves systemic vasoconstriction.
Additionally, serotonin is stored in blood platelets andis released during agitation and vasoconstriction, where it then acts as an agonist to other platelets.
Like other local anesthetics(such as mepivacaine, and prilocaine), procaine is a vasodilator,thus is often coadministered with epinephrine for the purpose of vasoconstriction.
In general, when ligands bind to α-adrenergic receptors, vasoconstriction occurs, and when binding to β1-adrenergic receptors, cardiovascular contractility and increased heart rate result.
These activities increase cardiac output and increase blood flow to peripheral vascular beds.[1][3] It is not an α-adrenergic agonist,does not cause vasoconstriction, and is not a pressor agent.[2].
ROCK induces vasoconstriction by phosphorylating the myosin-binding subunit of myosin light chain(MLC) phosphatase, thus decreasing MLC phosphatase activity and enhancing vascular smooth muscle contraction.[5].
Thromboxane A2(TXA2) is generated in the lungs of people with asthma,and when it signals through the thromboxane receptor it causes bronchoconstriction, vasoconstriction, mucous secretion, and airway hyper-responsiveness.
Thanks to this characteristic, phenylephrine is able to induce vasoconstriction by exercising- depending on the route through which it is administered- an anti-hypotensive action, a mydriatic action, or a decongestant action at the level of the nasal mucosa.
Rizatriptan acts as an agonist at serotonin 5-HT1B and 5-HT1D receptors.[8] Like the other triptans sumatriptan and zolmitriptan,rizatriptan induces vasoconstriction- possibly by inhibiting the release of calcitonin gene-related peptide from sensory neurons in the trigeminal nerve.[8].
In addition to causing vasoconstriction, experimental data from animal studies show that Zolmitriptan also activates 5-HT1 receptors on peripheral terminals of the trigeminal nerve innervating cranial blood vessels, which may also contribute to the antimigrainous effect of Zolmitriptan in humans.
It has also been used(in conjunction with sodium nitroprusside) successfully as an antidote to reverse the severe peripheral vasoconstriction which can occur as a result of overdose with certain 5-HT2A agonist drugs such as 25I-NBOMe,[citation needed] DOB and Bromodragonfly(prolonged severe vasoconstriction can lead to gangrene if untreated).[1][2].