Примеры использования Membrane potential на Английском языке и их переводы на Русский язык
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It is these ions that alter the membrane potential.
This membrane potential is necessary for the TIM-mediated protein import.
For instance, oxidants can activate uncoupling proteins that reduce membrane potential.
Therefore, the presence of the membrane potential is essential for the life of the cell.
When membrane potential is lost during apoptosis, the concentration of phosphonium cations in a cell decreases.
Deletions to this protein causes swelling,a loss of membrane potential, and mitochondrial fragmentation.
Membrane potential monitoring can give the information about the kinetics of apoptotic processes in the tissues under study 8.
As a result of ion concentrations on both sides of the membrane are aligned, the membrane potential disappears and the cell dies.
As the membrane potential is increased, sodium ion channels open, allowing the entry of sodium ions into the cell.
The gradient also provides control of the concentration of ions such as Ca2+ driven by the mitochondrial membrane potential.
The direct current also leads to a change in the membrane potential through the changes in the electrolyte areas around the cell and in the cell.
In 2007, 18 F-labeled phosphonium cation( 18 F-FBnTP)was used for noninvasive study of mitochondrial membrane potential by PET 94.
With respect to the exterior of the cell,typical values of membrane potential, normally given in millivolts, range from -40 mV to -80 mV.
Because of this, there is a potential difference between the inner andouter surfaces of the cell membrane- the membrane potential.
Membrane potential is maintained principally by the concentration gradient and membrane permeability to potassium with some contribution from the Na+/K+ pump.
The gradient of chloride is reversed in immature neurons, andits reversal potential is higher than the resting membrane potential of the cell;
Increased extracellular potassium levels result in depolarisation of the membrane potentials of cells due to the increase in the equilibrium potential of potassium.
It is thought that this current may play an important role in regulating neuronal activity,by helping to stabilize the resting membrane potential of the cell.
Thus when the membrane potential of one cell changes, ions may move through from one cell to the next, carrying positive charge with them and depolarizing the postsynaptic cell.
Those more"typical" potassium channels preferentially carry outward(rather than inward)potassium currents at depolarized membrane potentials, and may be thought of as"outwardly rectifying.
This temporary depolarization of postsynaptic membrane potential, caused by the flow of positively charged ions into the postsynaptic cell, is a result of opening ligand-gated ion channels.
Affects respiration/electron transfer: acts as an uncoupler, collapsing Proton Motive Force(PMF):pH gradient(ΔpH) and membrane potential(ΔΨ) in bacterial systems, thereby reducing ATP synthesis.
Another hypothesis for respirasome formation is that membrane potential may initiate changes in the electrostatic/hydrophobic interactions mediating the assembly/disassembly of supercomplexes.
Tandem pore domain potassium channel- are constitutively open or possess high basal activation,such as the"resting potassium channels" or"leak channels" that set the negative membrane potential of neurons.
In non-excitable cells, and in excitable cells in their baseline states, the membrane potential is held at a relatively stable value, called the resting potential. .
The damage of mitochondrial membrane potential Δψ m and permeability of mitochondrial membrane can result from an oxidative stress or chemotherapy leading to the release of membrane-associated proteins including cytochrome c 91, 92.
Compared to the inner membranes of mitochondria,which have a significantly higher membrane potential due to charge separation, thylakoid membranes lack a charge gradient.
At membrane potentials negative to potassium's reversal potential, inwardly rectifying K+ channels support the flow of positively charged K+ ions into the cell,pushing the membrane potential back to the resting potential. .
The outward currents gene, respectively contribute to repolarization of cardiac action potentials, which helps bring membrane potentials back to the resting level and terminate a ventricular contraction.
Deletion of the junction protein MIC60/Mic60(formerly Mitofilin/Fcj1)leads to a reduced inner membrane potential and impaired growth and to dramatically aberrant inner membrane structures which form concentric stacks instead of the typical invaginations.