Примери за използване на Active in vitro на Английски и техните преводи на Български
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Emtricitabine is active in vitro against HBV.
At least one metabolite of MMAE has been shown to be active in vitro.
Cidofovir is active in vitro against HCMV, a member of the herpesviridae family.
The predominant biotransformation product(albumin bound)was minimally active in vitro.
Antiviral activity Cidofovir is active in vitro against HCMV, a member of the herpesviridae family.
Additionally, velpatasvir is active in vitro against most of the NS5A RAVs that confer resistance to ledipasvir and daclatasvir.
Clarithromycin is suitable for initial treatment of community-acquired respiratory tract infections(out-hospital),and is active in vitro against the most common and atypical respiratory pathogens.
Voxilaprevir is active in vitro against most of the NS3 RAVs that confer resistance to first generation NS3/4A protease inhibitors.
Elbasvir is active in vitro against genotype 1a NS5A substitutions, M28V and Q30L, genotype 1b substitutions, L28M/V, R30Q, L31V, Y93C, and genotype 4 substitution, M31V, which confer resistance to other NS5A inhibitors.
The main metabolite in plasma, the glucuronide conjugate of silodosin(KMD-3213G),that has been shown to be active in vitro, has an extended half-life(approximately 24 hours) and reaches plasma concentrations approximately four times higher than those of silodosin.
Due to its novel viral target enfuvirtide is equally active in vitro against both wildtype laboratory and clinical isolates and those with resistance to 1, 2 or 3 other classes of antiretrovirals(nucleoside reverse transcriptase inhibitors, non-nucleoside reverse transcriptase inhibitors and protease inhibitors).
Ciprofloxacin has been shown to be active in vitro against most strains of the following organisms, however, the clinical significance of these data is unknown.
Loperamide caused a< 30% decrease in the formation of telotristat(active metabolite) in vitro(see section 5.2).
Microbiology Posaconazole has been shown in vitro to be active against the following microorganisms.
Cefuroxime is usually active against the following microorganisms in vitro.
In vitro, dasatinib is active in leukaemic cell lines representing variants of imatinib-sensitive and resistant disease.
Cefuroxime is active against the following organisms in vitro.
At both week 24 andweek 120, responder rates were higher for patients on 3 or more active substances(in vitro) in their background regimen.
The major metabolite of ticagrelor is AR-C124910XX,which is also active as assessed by in vitro binding to the platelet P2Y12 ADP-receptor.
Liposomal-encapsulated compared with conventional doxorubicin was not found more active in doxorubicin resistant cell lines in vitro.
In vitro telotristat(active metabolite) caused a concentration dependent increase in CYP2B6 mRNA levels(> 2-fold increase and> 20% of the positive control, with a maximum observed effect similar to the positive control), suggesting possible CYP2B6 induction(see section 4.5).
The active thiol metabolite which has been isolated in vitro, binds rapidly and irreversibly to platelet receptors, thus inhibiting platelet aggregation.
In vitro clavulanic acid and amoxicillin in combination are active against a wide range of clinically important bacteria including the following organisms which are commonly associated with bovine mastitis.
In vitro and in vivo losartan and its pharmacologically active carboxylic acid metabolite E-3174 block all physiologically relevant actions of angiotensin II, regardless of the source or route of its synthesis.
In vitro and in vivo losartan and its pharmacologically active carboxylic acid metabolite E-3174 block all physiologically relevant actions of angiotensin II, regardless of the source or route of its synthesis.
In vitro and in vivo losartan and its pharmacologically active carboxylic acid metabolite E-3174 block all physiologically relevant actions of angiotensin II, regardless of the source or route of its synthesis.
Mechanism of action and pharmacodynamic effects The active substance accelerated re-epithelialization in an in vitro wound scratch assay using human primary keratinocytes at the dosage of 1 μg/ml, and in a porcine ex vivo wound healing model at the dosage of 10 µg/ml.