Примери за използване на In vitro studies have shown на Английски и техните преводи на Български
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In vitro studies have shown that montelukast is a potent inhibitor of CYP 2C8.
Active substances that may increase gefitinib plasma concentrations In vitro studies have shown that gefitinib is a substrate of p-glycoprotein(Pgp).
In vitro studies have shown that paliperidone is a P-glycoprotein(P-gp) substrate.
Desloratadine does not inhibit CYP3A4 in vivo, and in vitro studies have shown that the medicinal product does not inhibit CYP2D6 and is neither a substrate nor an inhibitor of P-glycoprotein.
In vitro studies have shown potential interaction of patiromer with quinidine.
Furthermore in vitro studies have shown that ospemifene is a weak inducer for CYP2B6 and CYP3A4.
In vitro studies have shown that paliperidone is a P-glycoprotein(P-gp) substrate.
Additionally, in vitro studies have shown that saquinavir is a substrate and an inhibitor for P-glycoprotein(P-gp).
In vitro studies have shown that saquinavir is a substrate for P-glycoprotein(P-gp).
Additionally, in vitro studies have shown that saquinavir is a substrate and an inhibitor for P-glycoprotein(P-gp).
In vitro studies have shown that osimertinib is not a substrate of OATP1B1 and OATP1B3.
In vitro studies have shown that degarelix is not a substrate for the human CYP450 system.
In vitro studies have shown that gefitinib has limited potential to inhibit CYP2D6.
In vitro studies have shown that cannabinoids modulate immune cells, such as T cells and macrophages.
In vitro studies have shown some potential for celecoxib to inhibit CYP2C19 catalysed metabolism.
In vitro studies have shown that cabazitaxel is mainly metabolised through CYP3A(80% to 90%)(see section 5.2).
In vitro studies have shown that CYP3A4 is the major enzyme involved in the metabolism of ketoconazole.
In vitro studies have shown that approximately 97.5% of circulating Amlodipine is bound to plasma proteins.
In vitro studies have shown that aclidinium or its metabolites are not substrates or inhibitors of P-glycoprotein.
In vitro studies have shown that paliperidone is a P-gp substrate and a weak inhibitor of P-gp at high concentrations.
In vitro studies have shown that brivaracetam exhibits little or no inhibition of CYP450 isoforms except for CYP2C19.
In vitro studies have shown that tivozanib is neither a substrate nor an inhibitor of the multidrug efflux pump, P-glycoprotein.
In vitro studies have shown that ketoconazole may inhibit the metabolism of granisetron via the cytochrome P450 3A isoenzyme family.
In vitro studies have shown that dimethyl fumarate at a therapeutic dose does not inhibit CYP3A4/5 and BCRP and is a weak P-glycoprotein inhibitor.
Preliminary in vitro studies have shown that safinamide is not a substrate for the transporters P-gp, BCRP, OAT1B1, OAT1B3, OATP1A2 or OAT2P1.
In vitro studies have shown that aclidinium bromide or the metabolites of aclidinium bromide are not substrates or inhibitors of P-glycoprotein.
In vitro studies have shown the increase in repaglinide exposure is due to inhibition of CYP2C8 by the glucuronide metabolite of clopidogrel.
In vitro studies have shown that tivozanib is neither a substrate nor inhibitor of the transporter proteins MDR1(P-gp), OCT1, OATP1B1, OATP1B3 and BSEP.
In vitro studies have shown that drug interactions between tedizolid and inhibitors or inducers of cytochrome P450(CYP) isoenzymes are unanticipated.