Примери за използване на Potassium channels на Английски и техните преводи на Български
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Trobalt acts by keeping the potassium channels open.
KCNQ5, together with other potassium channels including KCNQ1 and KCNQ4, functions in vascular smooth muscle.
This means that it opens specific pores on nerve cells called potassium channels.
This process requires energy-driven potassium channels to move charge against a gradient.
CB1 receptors are also coupled through G proteins to several types of calcium and potassium channels.
Trobalt has an effect on the potassium channels located on the nerve cells of the brain.
This means that it opens specific pores on the surface of nerve cells called potassium channels.
Physiologically, the potassium channels of hair follicles are regulated by intracellular levels of nucleotides(e.g., ATP and ADP).
No pharmacological activity was found for the fampridine metabolites against selected potassium channels in vitro.
Although the metabolite is inactive at potassium channels, potential off target effects due to accumulation are unknown.
Sulphonylureas will act on pancreatic beta-cells by inhibiting ATP-sensitive potassium channels.
Amifampridine blocks voltage-dependent potassium channels, thereby prolonging pre-synaptic cell membrane depolarisation.
Delamanid and/or its metabolites have the potential to affect cardiac repolarisation via blockade of hERG potassium channels.
This captured energy revitalizes potassium channels and is necessary for the regulation of ion transport across the cellular membranes.
Scientists have demonstrated that as people experience hair loss the function of potassium channels within hair follicles diminishes.
Repaglinide closes ATP-dependent potassium channels in the β-cell membrane via a target protein different from other secretagogues.
This interaction of glimepiride from communicating with him protein regulates the opening orclosing of ATP-sensitive potassium channels.
Repaglinide closes ATP-dependent potassium channels in the β-cell membrane via a target protein different from other secretagogues.
Like all other derivatives sulfonylureas,glimepiride regulates insulin secretion by interacting with ATP-sensitive potassium channels in membranes?-cells.
Without sufficient calcium, potassium channels that can't be opened and closed properly, resulting in impaired nerve signaling.
A study in cloned human cardiac ion channels has shown that granisetron has the potential to affect cardiac repolarisation via blockade of hERG potassium channels.
Potassium channels are one of the voltage-gated ion channels found in neuronal cells and are important determinants of neuronal activity.
Granisetron has been shown to block both sodium and potassium channels, which could affect cardiac depolorisation and repolarisation and therefore PR, QRS, and QT intervals.
Potassium channels are small pore-forming protein structures that control the transport of nutrients across the hair follicle's cell membrane.
Nicorandil has dual pharmacological effects;activation of ATP-sensitive inward-rectifier potassium channels and(similar to nitroglycerin) increased production of nitric oxide.
Nateglinide closes ATP-dependent potassium channels in the beta-cell membrane with characteristics that distinguish it from other sulphonylurea receptor ligands.
However, as a known potent potassium channel opener,it can be assumed that the drug targets the same protein structures(potassium channels) as TRX2® does.
This leads to the closure of ATP-dependent potassium channels and the depolarization of the cell membrane, which in turn facilitates the opening of calcium channels. .
A study in cloned human cardiac ion channels has shown that granisetron has the potential to affect cardiac repolarisation via blockade of hERG potassium channels.
Closure of the potassium channels leads to depolarisation of the beta cell and results by opening of the calcium channels- to an increased influx of calcium into the cell.