Примери коришћења Platelet на Енглеском и њихови преводи на Српски
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Platelet consumption increases with DIC.
No, the patient has a platelet disorder.
Platelet formation decreases with malnutrition.
Lacking the factor for platelet adhesion.- You're doing bleeding time?
Platelet survival is normal but function is not.
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Some patients may have spontaneous platelet aggregation.
Platelet like blood transfusion, but in this case it.
It does not inhibit platelet aggregation, where PGI2 does.
Platelet and red blood cell counts were beginning to hold.
Also characterized by defective platelet granule release, and as.
When platelet reduction has no direct connection with any.
Glycoprotein lb, which determines the platelet adhesion factor.
Platelet interaction with heparin. Communicate it to the surface.
Adrenaline. The presence in the blood smear giant platelet yields.
Platelet pathology is the causebleeding in nearly 80% of cases.
The translation has no effect on platelet. Here the text of the cover.
The platelet dysfunction and cardiac arrest points to a pancreatic tumor.
Using TEG®, it is also possible to evaluate the degree of platelet inhibition using arachidonic acid and adenosine diphosphiate.
Your platelet level is 10 times normal and you have very low levels of factor 8.
At the primary level of thrombopoietin thrombocytosisincreased or normal, andmay have abnormalities in platelet maturation processes.
And the extent of platelet activation caused by this interaction.
It is an antioxidant phenol and a potent vasodilator that inhibits serum triglyceride synthesis,lipid peroxidation, and platelet aggregation.
Granules and platelet thrombocytopathy. Thrombocytopenia, combined with.
These vascular effects lead to thickening of blood vessel walls andenhance blood platelet stickiness, increasing the likelihood that clots will form and trigger heart attacks and strokes.
It also affects platelet aggregation but the relevance of this effect to the treatment of pulmonary hypertension is unknown.
Utilized by LPA for cell signaling, EDG receptors mediate diverse biologic functions,including proliferation, platelet aggregation, smooth muscle contraction, inhibition of neuroblastoma cell differentiation, chemotaxis, and tumor cell invasion.
An antiplatelet drug(antiaggregant) is a member of a class of pharmaceuticals that decrease platelet aggregation and inhibit thrombus formation.
It itself is not involved in platelet activation and aggregation in case of a wound, but its precursor, thromboxane A2.
It blocks serotonin-induced platelet aggregation, and has applications in the treatment of many diseases including diabetes mellitus, Buerger's disease, Raynaud's disease, coronary artery disease, angina pectoris, and atherosclerosis.
Amplification of platelet phagocytosis by macrophages system and stimulate.