Examples of using Stroke volume in English and their translations into Portuguese
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SVV- stroke volume variation.
Increased left ventricular output and increased stroke volume.
SBP is a function of HR, stroke volume, contractility, preload and afterload.
The DC each ventricle was determined by the equation:DC stroke volume* FCF.
Therefore, stroke volume, cardiac output, and blood pressure are reduced.
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Decreased venous return has the opposite effect,causing a reduction in stroke volume.
Stroke volume was determined echocardiographically by Simpson's modified biplane method.
The sympathetic innervation of the heart controls the HR,ventricular contractility, and stroke volume.
Stroke Volume(SV): volume of blood pumped from the left ventricle per heartbeat.
Elevated afterload(commonly measured as the aortic pressure during systole)reduces stroke volume.
Stroke volume results from the difference between IV systolic and end-diastolic volume. .
According to the Fick principle,VO2 varies with HR, stroke volume, and arteriovenous oxygen difference.
Tachycardia, increased stroke volume and cardiac output CO occur as compensatory mechanisms to vasodilation.
A study on dogs showed a decrease in cardiac output and stroke volume of 36% after IAP increased to 40 mm Hg.
In fact, stroke volume increased, which suggests an optimization of cardiac efficiency and myocardial oxygen utilization.
Caffeine has been shown to increase heart rate,left ventricular output, and stroke volume in published studies.
Data regarding heart rate, stroke volume, cardiac output and peripheral vascular resistance are shown in Table 3.
This higher heart rate observed in pediatric population is a compensatory mechanism for their smaller hearts and lower stroke volume.
Cardiac stroke volume is in direct proportion with heart rate, haemoglobin level and arterial blood oxygen content.
Prolonged aerobic exercise training may also increase stroke volume, which frequently results in a lower(resting) heart rate.
Stroke volume measurements based on MRI and TTE showed a moderate correlation r=0.56, p.
To calculate percent volume responsiveness,cardiac output or stroke volume must be known in advance and after these maneuvers.
However, stroke volume depends on several factors such as heart size, contractility, duration of contraction, preload(end-diastolic volume), and afterload.
However, after having reached 110-120 beats per minute, the stroke volume stabilizes, and HR alone contributes to increased cardiac output.
An increase in the volume or speed of venous return will increase preload and, through the Frank-Starling law of the heart,will increase stroke volume.
In cardiovascular physiology, stroke volume(SV) is the volume of blood pumped from the left ventricle per beat.
The different etiologies can be divided into groups, with variations according to the form of the disease, such as the presence orabsence of symptoms, stroke volume and composition of the liquid.
Advanced hemodynamic parameters such as stroke volume(SV) and stroke volume variation(SVV), are key to optimal fluid administration.
The stroke volume of each ventricle was determined by the formula: stroke volume ventricular volume at end diastole- ventricular volume at end systole.
Intravenous etilefrine increases the pulse rate,cardiac output, stroke volume, central venous pressure and mean arterial pressure of healthy individuals.