Examples of using Viral replication in English and their translations into Spanish
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Official
How it exactly affects viral replication is unknown;
In this form it interferes with RNA metabolism required for viral replication.
This stage of viral replication can be inhibited in two ways.
The drugs inhibit this enzyme and prevent the viral replication.
The small HDAg activates the viral replication joining directly to the RNA.
Thus, it helps fight against some bacteria and inhibit viral replication.
Viral replication takes place in tissues as diverse(lymph node, intestine, brain, thymus,…).
Recombinant interferon alpha-2also inhibits viral replication in vitro and in vivo.
Viral replication then leads to viremia and the virus spreads to its secondary site of infection, the central nervous system CNS.
Shown to thwart viral infection& block viral replication.
Depending on thespecies of the virus, infection and viral replication may result in host cell lysis and formation of a viral plaque.
Vif is a 23-kilodalton protein that is essential for viral replication.
This technique stimulates the immune system preventing viral replication in animals, therefore decreases the viral circulation.
Another explanation could be the presence of cellular enzymes required for viral replication.
Interferons are named for their ability to"interfere" with viral replication by protecting cells from virus infections.
It recognizes and cuts all known types of the hepatitis C virus,thereby stopping viral replication.
High potency vaccines and/or multiple vaccinations may be expected to reduce viral replication, the frequency of the carrier state and NSP seroconversion.
The viruses HIV andhepatitis B can persist in C. lectularius for two weeks, but with no viral replication.
Nevertheless, the virus persists elsewhere, particularly in lymph nodes and here viral replication continues as follicular dendritic cells interact with more CD4+ cells that become infected.
In addition, it attracts and grabs hold of viral particles,thus helping to stop viral replication.
Right now, all of the approved antivirals block viral replication at the same site;
However, this is not always possible as many patients with active viral replication are asymptomatic.
Foreign, bacterial DNA is sometimes misincorporated in the capsid head of bacteriophage virus particles as DNA is packaged into new bacteriophages during viral replication.
This compound directly inhibits the virus by interfering with viral replication mechanisms.
Some of these new drugs exert their effect by directly inhibiting some proper mechanism of virus replication, are called"direct inhibitors" of viral replication.
Absence of antibodies does not imply susceptibility since cellular immunity controls viral replication.
However, improving the effectiveness of therapeutic vaccines against HIV requires, among other things,a better understanding of the immune mechanisms of control of viral replication.
By the early 1980s, genes for these interferons had been cloned,adding further definitive proof that interferons were responsible for interfering with viral replication.
It has been found higher viral load in patients with fevers, bronchopneumonia, idiopathic inflammatory myopathies intense,patients with chronic lymphocytic leukemia and polycythemia vera, periodontitis in gingiva, etc., as an indication that inflammatory processes facilitate viral replication.
