Examples of using Catecholamines in English and their translations into Vietnamese
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Catecholamines are synthesized from tyrosine.
The latter three are known as the catecholamines.
The catecholamines travel through the blood and bind to receptors on fat cells.
Caffeine is a stimulant, which causes your body to release catecholamines.
Catecholamines have a half-life of approximately a few minutes when circulating in the blood.
Dopexamine may potentiate the effects of other catecholamines like noradrenaline.
It helps the catecholamines stick around longer, it doesn't increase their production outright.
If available,treatment with angiotensin II infusion and/or intravenous catecholamines may also be considered.
The catecholamines(epinephrine, norepinephrine, and dopamine) are found in the adrenal medulla, neurons, and brain.
They are chemically related to the human body's natural catecholamines and epinephrine and norepinephrine.
The body's primary method of stimulating lypolisis is the production of adrenaline or noradrenalin,which are known as catecholamines.
With excess of glucocorticoids, catecholamines, or glucagon, increased hepatic output of glucose is a contributory factor;
To release fatty acids from fat cells so they can be burned for energy,your body produces chemicals called catecholamines.
Metyrosine prevents the tumor from making certain natural substances(catecholamines) that raise the blood pressure and make the heart beat faster.
Just having catecholamines is good enough to prevent many side effects of stress and, hopefully, you can escape the threat before they get depleted.
Such a decrease in blood pressure is not accompanied by marked changes in the heartrate or the level of catecholamines in blood plasma with prolonged use.
Catecholamines act as both hormones and neurotransmitters, and drugs that mimic them are typically used to treat low blood pressure and cardiac arrest.
L-DOPA is the precursor to the neurotransmitters dopamine, norepinephrine(noradrenaline), and epinephrine(adrenaline),which are collectively known as catecholamines.
All of the early β2 agonist catecholamines used for bronchospasm had strong side effects, with increase in heart rate as the most common and most problematic.
This process is mediated by insulin and blood-borne fatty acids; when levels drop,an energy deficit is“sensed” and catecholamines(adrenaline and nordrenaline) increase.
Normally, catecholamines and their metabolites are present in the body in small, fluctuating amounts that only increase appreciably during and shortly after a stressful situation.
Levodopa can influence the results of laboratory determination of creatinine, catecholamines, glucose and uric acid, and a false-positive result of the Coombs test is also possible.
In the case of catecholamines, decreased insulin release is an additional factor in producing carbohydrate intolerance, and with excess somatostatin production it is the major factor.
It inhibits the enzyme tyrosine hydroxylase and, therefore, catecholamine synthesis, which, as a consequence, depletes the levels of the catecholamines dopamine, adrenaline and noradrenaline in the body.
It's chemically similar to ephedrine and catecholamines(the chemicals adrenaline and noradrenaline, which cause the breakdown of fat cells), and although less potent than those two, it induces similar effects.
If the person has not had a recent episode of hypertension,their blood and urine concentrations of catecholamines could be at normal or near normal levels even when a pheochromocytoma is present.
Unlike caffeine, for example, which loses its fat-burning mojo the longer you use it, ephedrine's effects are amplified by continueduse because it appears to increase your body's sensitivity to catecholamines.
However, it is known that this drug has important implications for catecholamines, serotonin, glutamate, amino-butyric acid gamma, orexin and histamine systems in the brain.
A report published in the Indian Journal of Endocrinology and Metabolism states that,“Stress can lead to changes in theserum level of many hormones including glucocorticoids, catecholamines, growth hormone and prolactin.”.
The primary endogenousagonists of the sympathetic nervous system are the catecholamines(i.e., epinephrine[adrenaline], norepinephrine[noradrenaline], and dopamine), which function as both neurotransmitters and hormones.