Examples of using Randomisation in English and their translations into Slovenian
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Colloquial
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Official
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Medicine
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Ecclesiastic
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Financial
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Computer
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Official/political
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Programming
Weeks after randomisation.
IIa: at least one well-designed controlled study without randomisation.
Months from Randomisation.
In all patients,assessment of efficacy was 8 weeks after the randomisation.
Months since randomisation.
All patients were followed-up until withdrawal orup to 3 years after randomisation.
Time since randomisation(week).
Randomisation was stratified by HCV genotype(1, 2, 3, 4, 5, 6 and indeterminate).
IIa Evidence obtained from atleast one well designed controlled study without randomisation.
Time since Randomisation(Week) IDegLira=Xultophy.
Includes patients who received atleast one dose of blinded medicinal product after randomisation.
Randomisation was stratified by screening HIV-1 RNA level(≤ 100,000 copies/ml orgt; 100,000 copies/ml).
Post-hoc efficacy analysis in patients whomaintained a high or very high DRL at randomisation.
At randomisation, 70% of patients were NYHA class II, 24% were class III and 0.7% were class IV.
The primary outcome measurewas overall survival defined as the time from randomisation until death from any cause.
Randomisation was stratified by HCV genotype(1, 2, 4, 6, and indeterminate) and the presence or absence of cirrhosis.
During SET, patient diaries were recorded for an average of 88 days during screening and133 days during randomisation.
Randomisation was stratified by BRAF and tumour PD-L1 status and best response to prior ipilimumab.
The median duration of time from initial diagnosis to randomisation was 2.6 years in both the nivolumab and everolimus groups.
Randomisation was followed by a 12-week Natpar titration phase and a 12-week Natpar dose maintenance phase.
Tumour assessments were conducted 12 weeks after randomisation then every 6 weeks for the first year, and every 12 weeks thereafter.
Randomisation was stratified by prior trastuzumab treatment and number of prior treatments for metastatic disease.
The primary efficacy parameter of the trial was overall survival,defined as the time from randomisation to death from any cause.
Randomisation was stratified by sites of disease and by time from discontinuation of prior adjuvant anti-oestrogen therapy.
A significant reduction in the number of HDDs andTAC occurred in some patients in the period between the initial visit(screening) and randomisation due to non-pharmacological effects.
Randomisation was stratified by breast cancer type(operable, locally advanced, or inflammatory) and ER and/or PgR positivity.
PFS was defined as the time from randomisation to the earliest date of disease progression or death from any cause, or to the date of censor.
At randomisation, these patients consumed such a small amount of alcohol that there was little room for further improvement(floor effect).
At randomisation, active forms of vitamin D were reduced by 50% and patients were allocated to Natpar 50 micrograms daily or placebo.
Randomisation was stratified by geographic region, time to progression from the start of first-line therapy(< 6 months versus≥6 months) and disease measurability.