Examples of using Codon in English and their translations into Slovenian
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Medicine
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Colloquial
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Official
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Ecclesiastic
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Financial
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Computer
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Official/political
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Programming
What is the stop codon?
The methionine codon AUG is also the most common start codon.
P53 gene mutation and codon 55.
The translation code from RNA codon to amino acid is the same for most organisms.
You may find more data at codon.
Mutations at protease codons 33, 82, 84 and 90:.
It is therefore possible to have 64 different codons.
The double helix has an alphabet of 64 codons, that are used to encode our human DNA.
Each codon encodes for a specific amino acid, except the stop codons, which terminate protein synthesis.
Additional mutations in KRAS and NRAS at exon 3(codon 59) were subsequently identified(n= 7).
Each codon encodes a particular amino acid, with the exception of the stop codon, which stops protein synthesis.
All tested cases of vCJD todate have been MM homozygotes at codon 129 of the prion protein gene.
The translation code from RNA codon to amino acid is the same for most organisms, but slightly different for some.
Eighteen bases coding six histidines are inserted right after the START codon or right before the STOP codon.
Mutations of the KRAS gene at certain hot-spots(mainly codons 12 and 13) result in constitutive activation of the KRAS protein independently of EGFR signalling.
Over 95% of cases of MEN 2A areassociated with mutations in the ret proto-oncogene affecting 1 of 5 codons in exon 10.
Lysine(abbreviated as Lys or K),[1] encoded by the codons AAA and AAG, is an α-amino acid that is used in the biosynthesis of proteins.
Have you considered using plasmids totransfect the new base-pair vectors directly into the codon sequence, so that won't happen?
(t) Estimated from 16,403 melanomas with annotated BRAF codon 600 mutations in the public COSMIC database, release 71(November 2014).
Preclinical data generated in biochemical assays demonstrated thatdabrafenib inhibits BRAF kinases with activating codon 600 mutations(Table 5).
Resistance: HIV-1 resistance to emtricitabine develops as the result of changes at codon 184 causing the methionine to be changed to a valine(an isoleucine intermediate has also been observed) of the HIV reverse transcriptase.
Preclinical data generated in biochemical assays demonstrated that vemurafenib canpotently inhibit BRAF kinases with activating codon 600 mutations(table 6).
Relationships between baseline phenotypic susceptibility to tipranavir, primary PI mutations,protease mutations at codons 33, 82, 84 and 90, tipranavir resistance-associated mutations, and response to APTIVUS/ ritonavir therapy were assessed.
With respect to mutations on codons 33, 82, 84 and 90 approximately 4% had no mutations, 24% had mutations at codons 82(less than 1% of patients had the mutation V82L) and 90, 18% had mutations at codons 84 and 90 and 53% had at least one key mutation at codon 90.
A reduced virological response was observed in patients with viral strains harbouring two or more mutations at HIV protease codons 33, 82, 84 or 90, and not receiving new enfuvirtide.
Another technique is to perform a PCR with primers that have repetitive histidine codons(CAT or CAC)right next to the START or STOP codon in addition to several(16 or more) bases from one end of the DNA encoding the protein to be tagged(see primer example below).
Analyses were conducted to assess virological outcome by the number ofprimary PI mutations(any change at protease codons 30, 32, 36, 46, 47, 48, 50, 53, 54, 82, 84, 88 and 90) present at baseline.
Another technique is to perform a PCR with primers that have repetitive histidine codons(CAT or CAC)right next to the START or STOP codon in addition to several(16 or more) bases from one end of the DNA encoding the protein to be tagged(see primer example below).
Tumour samples obtained from the primary resection of colorectal cancer were analysed for thepresence of the seven most common activating mutations in the codon 12 and 13(Gly12Asp, Gly12Ala, Gly12Val, Gly12Ser, Gly12Arg, Gly12Cys, and Gly13Asp) of the KRAS gene by using an.
