Примери за използване на Viral replication на Английски и техните преводи на Български
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Viral replication is cytoplasmic.
Enzyme responsible for viral replication.
Viral replication is accompanied by widespread and severe focal necrosis.
Experience in patients with persistent viral replication.
The effect of Jakavi on viral replication in patients with chronic HBV infection is unknown.
It is NS5B polymerase inhibitor which is responsible for viral replication.
The increasing viral replication then triggers both humoral and cellular immunological responses.
Integrase is an HIV-1 encoded enzyme that is required for viral replication.
AZT and related nucleoside drugs inhibit viral replication by targeting specific viral enzymes.
Ombitasvir Ombitasvir is an inhibitor of HCV NS5A which is essential for viral replication.
By reducing viral replication and shedding, these medications slow the spread of infection in cells within the body.
Recombinant interferon alfa-2b also inhibits viral replication in vitro and in vivo.
Velpatasvir is a pan-genotypic HCV inhibitor targeting the HCV NS5A protein,which is required for viral replication.
Recombinant interferon alfa-2b also inhibits viral replication in vitro and in vivo.
Raltegravir inhibits the catalytic activity of integrase,an HIV-encoded enzyme that is required for viral replication.
It is defined as the presence of detectable viral replication for at least 6 months.
A microscopic, infectious organism that invades a living host andmakes copies of itself(viral replication).
A new HIV drug reduced viral replication and increased immune cells in individuals with advanced, drug-resistant HIV infection.
So far these antivirals have demonstrated at least a 90% reduction in viral replication.
A new HIV drug can reduce the viral replication and increase immune cells in individuals with advanced, drug-resistant HIV infection.
Clinical infection in human and nonhuman primates is associated with rapid and extensive viral replication in all tissues.
This action inhibits viral replication or if replication occurs, the progeny virions are unable to leave the cell.
Simeprevir is a specific inhibitor of the HCV NS3/4A serine protease,which is essential for viral replication.
Tipranavir is a non-peptidic inhibitor of the HIV-1 protease that inhibits viral replication by preventing the maturation of viral particles.
Transplantation recipients andpatients with advanced liver disease are at greater risk from active viral replication.
The inhibitory effect of fulvic acid is directed against the early stage of viral replication, so its preventive use is possible, in particular, during epidemics of influenza.
Sofosbuvir is a pan-genotypic inhibitor of the HCV NS5B RNA-dependent RNA polymerase,which is essential for viral replication.
All clinical trials recruited patients with CHB who had active viral replication measured by HBV DNA, elevated levels of ALT and a liver biopsy consistent with chronic hepatitis.
Exacerbations in patients with decompensated cirrhosis Transplantation recipients andpatients with decompensated cirrhosis are at greater risk from active viral replication.
Compensated liver disease andevidence of active viral replication, persistently elevated serum alanine aminotransferase(ALT) levels and histological evidence of active inflammation and/or fibrosis.