Examples of using Platelet function in English and their translations into Slovenian
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Platelet function is restored within 60 minutes of stopping infusion.
Medicinal products that inhibitserotonin uptake may lead to reduced platelet function.
Since iloprost inhibits platelet function its use with the following substances may enhance iloprost- mediated platelet inhibition, thereby increasing the risk of bleeding:.
Etoricoxib did not inhibit gastric prostaglandin synthesis andhad no effect on platelet function.
TP plays a role in platelet function; however, therapeutic doses of laropiprant had no clinically relevant effect on bleeding time and collagen-induced platelet aggregation(see section 4.5).
Nitric oxide: since both medicinal products inhibit platelet function, their combination may in.
Lumiracoxib at 800 mg daily did not lead to clinically meaningful inhibition of gastric prostaglandin synthesis andhad no effect on platelet function.
Platelet inhibition was readily reversed, with a return of platelet function towards baseline(gt; 50% platelet aggregation) 4 hours after stopping a continuous infusion of 2.0 microgram/ kg/ min.
Postoperative haemorrhage(P2Y12) is an inherited bleeding disorder in which platelet function is affected.
Medicinal products that affect coagulation or those that alter platelet function(e.g. ticlopidine, clopidogrel, LMWH) may increase the risk of bleeding prior to, during or after tenecteplase therapy.
Caution should be exercised if patients are required to take medicinal products that inhibit platelet function or anticoagulants.
Heparin, vitamin K antagonists and medicinal product that alter platelet function(such as acetylsalicylic acid, dipyridamole and abciximab) may increase the risk of bleeding if administered prior to, during or after reteplase therapy.
Patients were excluded from participation in SPRYCEL clinical trials if they took medicinal products that inhibit platelet function or anticoagulants.
Caution is advised in patients taking anticoagulantsand/or medicinal products known to affect platelet function[e.g. atypical antipsychotics and phenothiazines, most tricyclic antidepressants, non-steroidal anti-inflammatory drugs(NSAIDs), acetylsalicylic acid(ASA)](see section 4.5) and in patients with known bleeding tendencies/disorders.
If the patient requires semi-elective surgery,stop the eptifibatide infusion at an appropriate time to allow time for platelet function to return towards normal.
Iloprost can inhibit platelet function and its use with anticoagulants(such as heparin, coumarin-type anticoagulants) or other inhibitors of platelet aggregation(such as acetylsalicylic acid, non-steroidal anti-inflammatory medicinal products, ticlopidine, clopidogrel and glycoprotein IIb/ IIIa antagonists: abciximab, eptifibatide and tirofiban) may increase the risk of bleeding.
Caution is advised in patients taking anticoagulants and/ ormedicinal products known to affect platelet function, and in patients with known bleeding tendencies.
Pharmacogenetics: In patients who are poor CYP2C19 metabolisers, clopidogrel at recommended doses forms less of the active metabolite of clopidogrel andhas a smaller effect on platelet function.
Since platelets participate in the initiation and/or evolution of thrombotic complications of atherosclerotic disease,inhibition of platelet function can result in the reduction of the rate of cardiovascular events such as death, myocardial infarction, or stroke.
At the doses recommended for use in the treatment of essential thrombocythaemia, anagrelide may potentiate theeffects of other medicinal products that inhibit or modify platelet function e.g. acetylsalicylic acid.
As expected for a prostacyclin, iloprost produced haemodynamic effects(vasodilatation, reddening of skin, hypotension,inhibition of platelet function, respiratory distress) and general signs of intoxication such as apathy, gait disturbances, and postural changes.
Ticagrelor has been documented to augment the following adenosine-induced effects in healthy subjects and in patients with ACS: vasodilation(measured by coronary blood flow increases in healthy volunteers and ACS patients; headache),inhibition of platelet function(in human whole blood in vitro) and dyspnoea.
Consequently, platelets exposed to clopidogrel are affected for the remainder of their lifespan andrecovery of normal platelet function occurs at a rate consistent with platelet turnover.
Due to the irreversible binding, platelets exposed are affected for the remainder of their lifespan(approximately 7- 10 days)and recovery of normal platelet function occurs at a rate consistent with platelet turnover.
Acetylsalicylic acid and clopidogrel: In a clinical study in dyslipidaemic patients receiving both acetylsalicylic acid(81 mg) and clopidogrel(75 mg), laropiprant induced transient(4 hours post-dose)inhibition of platelet function in vivo(as evaluated by bleeding time and platelet aggregation studies), but had little effect across the dosing interval.
Puerarin has the function of inhibiting platelet aggregation, reducing blood viscosity and promoting blood micro-cycle.
This period is an average andreflects the time in which the entire population is reduced platelet cyclooxygenase functioning.
Your doctor may want to check your blood pressure,blood(platelet count) and the function of your kidneys.
Your doctor may want to check your blood pressure,blood(platelet count) and the function of your kidney.